PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
19360361-5	2009	In addition, z-IETD-fmk, a selective caspase-8 inhibitor, suppressed the nimesulide- and radiation-induced cleavage activation of caspase-9, caspase-3, caspase-8, and Bid, and suppressed the concomitant apoptosis, indicating that the nimesulide-induced increase in radiosensitivity was initiated by caspase-8.	nimesulide	73-83	BH3 interacting domain death agonist	Homo sapiens	167-170	
19360361-3	2009	The combined nimesulide-radiation treatment increased apoptosis, induced the cleavage of caspase-3, caspase-9, and poly(ADP-ribose) polymerase (PARP), activated caspase-8, and induced cleavage of Bid.	nimesulide	13-23	BH3 interacting domain death agonist	Homo sapiens	196-199	
16443156-5	2006	Nimesulide was administered for 4 weeks (10 mg/kg, ip, bid), at a dose equivalent to human therapeutic dosage.	nimesulide	0-10	BH3 interacting domain death agonist	Homo sapiens	55-58	
12439336-0	2002	Colocalization of BAX with BID and VDAC-1 in nimesulide-induced apoptosis of human colon adenocarcinoma COLO 205 cells.	nimesulide	45-55	BH3 interacting domain death agonist	Homo sapiens	27-30	
12439336-2	2002	In the present study the effect of nimesulide (NIM), a specific COX-2 inhibitor, on apoptosis and interactions between BCL-2 family death promoters BAX and BID and BAX and VDAC-1 were examined in human colon adenocarcinoma COLO 205 cells.	nimesulide	47-50	BH3 interacting domain death agonist	Homo sapiens	156-159