PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
27153185-0	2016	Glucose transporter type 10-lacking in arterial tortuosity syndrome-facilitates dehydroascorbic acid transport.	Dehydroascorbic Acid	80-100	solute carrier family 2 member 10	Homo sapiens	0-27	
27153185-2	2016	In this study GLUT10-mediated dehydroascorbic acid (DAA) transport was investigated, supposing its involvement in the pathomechanism.	Dehydroascorbic Acid	30-50	solute carrier family 2 member 10	Homo sapiens	14-20	
27153185-2	2016	In this study GLUT10-mediated dehydroascorbic acid (DAA) transport was investigated, supposing its involvement in the pathomechanism.	Dehydroascorbic Acid	52-55	solute carrier family 2 member 10	Homo sapiens	14-20	
27153185-4	2016	Silencing of GLUT10 decreased DAA transport in immortalized human fibroblasts whose plasma membrane was selectively permeabilized.	Dehydroascorbic Acid	30-33	solute carrier family 2 member 10	Homo sapiens	13-19	
27153185-6	2016	Re-expression of GLUT10 in patients" fibroblasts restored DAA transport activity.	Dehydroascorbic Acid	58-61	solute carrier family 2 member 10	Homo sapiens	17-23	
31621376-8	2021	GLUT10 was identified as an intracellular transporter of dehydroascorbic acid, which contributes to collagen and elastin crosslinking in the ER, redox homeostasis in the mitochondria and global and gene-specific methylation/hydroxymethylation affecting epigenetic regulation in the nucleus.	Dehydroascorbic Acid	57-77	solute carrier family 2 member 10	Homo sapiens	0-6	
30800210-2	2019	Transport of dehydroascorbic acid is impaired in the endomembranes of fibroblasts from arterial tortuosity syndrome (ATS) patients, due to the mutation in the gene coding for glucose transporter GLUT10.	Dehydroascorbic Acid	13-33	solute carrier family 2 member 10	Homo sapiens	195-201