PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 31621376-8 2021 GLUT10 was identified as an intracellular transporter of dehydroascorbic acid, which contributes to collagen and elastin crosslinking in the ER, redox homeostasis in the mitochondria and global and gene-specific methylation/hydroxymethylation affecting epigenetic regulation in the nucleus. Dehydroascorbic Acid 57-77 solute carrier family 2 member 10 Homo sapiens 0-6 30800210-2 2019 Transport of dehydroascorbic acid is impaired in the endomembranes of fibroblasts from arterial tortuosity syndrome (ATS) patients, due to the mutation in the gene coding for glucose transporter GLUT10. Dehydroascorbic Acid 13-33 solute carrier family 2 member 10 Homo sapiens 195-201 27153185-0 2016 Glucose transporter type 10-lacking in arterial tortuosity syndrome-facilitates dehydroascorbic acid transport. Dehydroascorbic Acid 80-100 solute carrier family 2 member 10 Homo sapiens 0-27 27153185-2 2016 In this study GLUT10-mediated dehydroascorbic acid (DAA) transport was investigated, supposing its involvement in the pathomechanism. Dehydroascorbic Acid 30-50 solute carrier family 2 member 10 Homo sapiens 14-20 27153185-2 2016 In this study GLUT10-mediated dehydroascorbic acid (DAA) transport was investigated, supposing its involvement in the pathomechanism. Dehydroascorbic Acid 52-55 solute carrier family 2 member 10 Homo sapiens 14-20 27153185-4 2016 Silencing of GLUT10 decreased DAA transport in immortalized human fibroblasts whose plasma membrane was selectively permeabilized. Dehydroascorbic Acid 30-33 solute carrier family 2 member 10 Homo sapiens 13-19 27153185-6 2016 Re-expression of GLUT10 in patients" fibroblasts restored DAA transport activity. Dehydroascorbic Acid 58-61 solute carrier family 2 member 10 Homo sapiens 17-23