PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
33554153-3	2020	We hypothesized that TRPC6 was essential in the maintenance or reexpression of a differentiated SMC phenotype and minimized luminal stenosis following arterial injury.	Phenobarbital	124-131	transient receptor potential cation channel, subfamily C, member 6	Mus musculus	21-26	
33554153-5	2020	At baseline, CCA of TRPC6-/- mice had reduced staining of MYH11 and SM22, fewer elastin lamina, luminal dilation, and wall thinning.	Phenobarbital	96-103	transient receptor potential cation channel, subfamily C, member 6	Mus musculus	20-25	
33554153-6	2020	After carotid wire injury, TRPC6-/- mice developed significantly more pronounced luminal stenosis compared with WT mice.	Phenobarbital	81-88	transient receptor potential cation channel, subfamily C, member 6	Mus musculus	27-32