PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
7667317-4	1995	Cells released from a mimosine block early in S phase stopped predominantly in G2/M in the presence of p53, confirming that p53 can mediate arrest at this stage, as well as in G1.	Mimosine	22-30	tumor protein p53	Homo sapiens	103-106	
9488455-5	1998	Unexpectedly, UVC treatment was also found to block the induction of p21Waf1 by various stress-inducing agents such as mimosine in the p53-deficient cells.	Mimosine	119-127	tumor protein p53	Homo sapiens	135-138	
9692554-3	1998	Using mimosine, a p21Waf1/Cip1 inducer that bypasses the requirement for transcriptional transactivation by p53, we demonstrated that a G1 cell cycle arrest can prevent apoptosis following UV-irradiation or treatment with an RNA polymerase 11 inhibitor.	Mimosine	6-14	tumor protein p53	Homo sapiens	108-111	
8809407-0	1996	p21WAF1/CIP1/SDI1 is elevated through a p53-independent pathway by mimosine.	Mimosine	67-75	tumor protein p53	Homo sapiens	40-43	
8809407-4	1996	In this study, we showed that mimosine can increase both p21 mRNA and protein levels, indirectly inhibit cyclin E-associated kinase activity without affecting the cyclin E protein level, block human breast cancer cells (21PT) in the late G1 phase of the cell cycle, and induce a p53-independent p21 pathway in these cells.	Mimosine	30-38	tumor protein p53	Homo sapiens	279-282	
7667317-4	1995	Cells released from a mimosine block early in S phase stopped predominantly in G2/M in the presence of p53, confirming that p53 can mediate arrest at this stage, as well as in G1.	Mimosine	22-30	tumor protein p53	Homo sapiens	124-127	
7530754-6	1994	While the p53 response was defective after radiation, agents that interfered with cell cycle progression such as mimosine, aphidicolin and deprivation of serum led to a normal p53 response in A-T cells.	Mimosine	113-121	tumor protein p53	Homo sapiens	176-179	
8247533-6	1993	Agents that produced double-strand breaks in DNA and/or inhibition of transcription caused an induction of p53 in the absence of radiation in control cells but not in ataxia-telangiectasia, but inhibitors of cell cycle progression such as mimosine and aphidicolin led to an increase in p53 in both cell types in the absence of radiation.	Mimosine	239-247	tumor protein p53	Homo sapiens	107-110