PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 9084984-0 1997 Activation of Na+/H(+)-exchanger by transforming Ha-ras requires stimulated cellular calcium influx and is associated with rearrangement of the actin cytoskeleton. Calcium 85-92 Harvey rat sarcoma virus oncogene Mus musculus 49-55 9084984-12 1997 The results show that both cytoskeletal rearrangement and activation of the Na+/H(+)-exchanger following expression of the Ha-ras oncogene require stimulated calcium influx and Cai oscillations. Calcium 158-165 Harvey rat sarcoma virus oncogene Mus musculus 123-129 7662118-4 1995 In the present studies, we measured the level and activity state of Ha-ras p21 protein in cultured keratinocytes undergoing calcium-induced differentiation. Calcium 124-131 Harvey rat sarcoma virus oncogene Mus musculus 68-74 1425589-6 1992 Active p21ras also partially replaces the requirement for protein kinase C activation in synergizing with a calcium ionophore to induce production of IL-2. Calcium 108-115 Harvey rat sarcoma virus oncogene Mus musculus 7-13 1503641-2 1992 Furthermore, initiated epidermal cells containing an activated Ha-ras oncogene have been shown to be resistant to calcium-induced terminal differentiation. Calcium 114-121 Harvey rat sarcoma virus oncogene Mus musculus 63-69 1503641-6 1992 Steady-state levels of Ha-ras mRNA remained unchanged in primary cultures of normal adult epidermal cells during calcium-induced differentiation, whereas steady-state levels of Ha-ras transcripts decreased during calcium-induced differentiation in primary newborn epidermal cells. Calcium 213-220 Harvey rat sarcoma virus oncogene Mus musculus 177-183