PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
1503641-2	1992	Furthermore, initiated epidermal cells containing an activated Ha-ras oncogene have been shown to be resistant to calcium-induced terminal differentiation.	Calcium	114-121	Harvey rat sarcoma virus oncogene	Mus musculus	63-69	
9084984-0	1997	Activation of Na+/H(+)-exchanger by transforming Ha-ras requires stimulated cellular calcium influx and is associated with rearrangement of the actin cytoskeleton.	Calcium	85-92	Harvey rat sarcoma virus oncogene	Mus musculus	49-55	
9084984-12	1997	The results show that both cytoskeletal rearrangement and activation of the Na+/H(+)-exchanger following expression of the Ha-ras oncogene require stimulated calcium influx and Cai oscillations.	Calcium	158-165	Harvey rat sarcoma virus oncogene	Mus musculus	123-129	
7662118-4	1995	In the present studies, we measured the level and activity state of Ha-ras p21 protein in cultured keratinocytes undergoing calcium-induced differentiation.	Calcium	124-131	Harvey rat sarcoma virus oncogene	Mus musculus	68-74	
1425589-6	1992	Active p21ras also partially replaces the requirement for protein kinase C activation in synergizing with a calcium ionophore to induce production of IL-2.	Calcium	108-115	Harvey rat sarcoma virus oncogene	Mus musculus	7-13	
1503641-6	1992	Steady-state levels of Ha-ras mRNA remained unchanged in primary cultures of normal adult epidermal cells during calcium-induced differentiation, whereas steady-state levels of Ha-ras transcripts decreased during calcium-induced differentiation in primary newborn epidermal cells.	Calcium	213-220	Harvey rat sarcoma virus oncogene	Mus musculus	177-183