PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28637791-0	2017	Store-operated calcium entry suppressed the TGF-beta1/Smad3 signaling pathway in glomerular mesangial cells.	Calcium	15-22	SMAD family member 3	Homo sapiens	54-59	
19468687-0	2009	Calcium signaling-induced Smad3 nuclear accumulation induces acetylcholinesterase transcription in apoptotic HeLa cells.	Calcium	0-7	SMAD family member 3	Homo sapiens	26-31	
19468687-3	2009	In this study, we have identified a potential Smad3/4 binding element, TGCCAGACA, located within the -601 to -571 bp fragment of the AChE promoter, as an important calcium response motif.	Calcium	164-171	SMAD family member 3	Homo sapiens	46-51	
19468687-7	2009	Calcium-induced AChE transcriptional activation was significantly blocked when the nuclear localization signal of Smad3 was destroyed.	Calcium	0-7	SMAD family member 3	Homo sapiens	114-119	
19468687-8	2009	Taken together, our data suggest Smad3 can regulate AChE transcriptional activation following calcium-induced nuclear accumulation.	Calcium	94-101	SMAD family member 3	Homo sapiens	33-38	
23996929-10	2014	Most importantly, we demonstrate that these effects of S100A2 are manifested through functional interaction with Smad3, which is enhanced in the presence of high calcium and TGF-beta.	Calcium	162-169	SMAD family member 3	Homo sapiens	113-118