PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
1660014-3	1991	In contrast, at concentrations that may be achieved in vivo following the administration of the drug (5-20 micrograms/ml), D impaired in a dose-dependent manner (p less than 0.05) the generation of O-2 in response to N-formyl-L-methionyl-L-leucyl-L-phenylalanine (FMLP, 1 microM) or calcium ionophore A23187 (10 microM).	Calcium	283-290	immunoglobulin kappa variable 1D-39	Homo sapiens	198-201	
7814447-9	1995	These results indicate that the effect of X-XO on calcium homeostasis appears to result from an interaction of O2- and H2O2, which could be explained by the formation of the hydroxyl radical.	Calcium	50-57	immunoglobulin kappa variable 1D-39	Homo sapiens	111-123	
6324816-5	1984	The rate and amount of O-2 generated by AGEPC-activated neutrophils were enhanced significantly in the presence of extra-cellular calcium.	Calcium	130-137	immunoglobulin kappa variable 1D-39	Homo sapiens	23-26	
6329209-4	1984	The amount of O-2 produced by ZAS stimulated neutrophils was enhanced in the presence of extra-cellular calcium.	Calcium	104-111	immunoglobulin kappa variable 1D-39	Homo sapiens	14-17	
6329209-5	1984	However, the intracellular calcium antagonist, 8-(N,N-diethylamino)-octyl-(3,4,5-trimethoxy) benzoate hydrochloride (TMB-8), caused a dose-related inhibition of ZAS -elicited O-2 production.	Calcium	27-34	immunoglobulin kappa variable 1D-39	Homo sapiens	175-178	
6090448-6	1984	Antagonists of calcium-binding proteins (i.e. phenothiazines, naphthalene sulfonamides) inhibited the release of O-2 in a fashion compatible with the involvement of calmodulin in these phenomena.	Calcium	15-22	immunoglobulin kappa variable 1D-39	Homo sapiens	113-116