PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34838920-3	2022	To elucidate the potential role of Wnt5a-mediated non-canonical Wnt-YAP/TAZ signaling in the lung injury induced by short-term exposure of benzo(a)pyrene (BaP, a representative PAHs), intratracheally instilled mouse model was used and further interfered with its Wnt5a level by small molecule antagonists and agonists.	Benzo(a)pyrene	155-158	wingless-type MMTV integration site family, member 5A	Mus musculus	35-40	
34838920-3	2022	To elucidate the potential role of Wnt5a-mediated non-canonical Wnt-YAP/TAZ signaling in the lung injury induced by short-term exposure of benzo(a)pyrene (BaP, a representative PAHs), intratracheally instilled mouse model was used and further interfered with its Wnt5a level by small molecule antagonists and agonists.	Benzo(a)pyrene	155-158	wingless-type MMTV integration site family, member 5A	Mus musculus	263-268	
34838920-6	2022	Functionally, inhibition of Wnt5a attenuated the BaP-induced inflammation and recuperated CC16 expression, as well as suppressed the epithelial cytokines release.	Benzo(a)pyrene	49-52	wingless-type MMTV integration site family, member 5A	Mus musculus	28-33	
34838920-8	2022	Our findings together suggest that Wnt5a is a potential endogenous regulator in lung inflammation and airway epithelial injury, and Wnt5a-YAP/TAZ signaling contributes to lung dysfunction in acute exposure to BaP.	Benzo(a)pyrene	209-212	wingless-type MMTV integration site family, member 5A	Mus musculus	35-40	
34838920-8	2022	Our findings together suggest that Wnt5a is a potential endogenous regulator in lung inflammation and airway epithelial injury, and Wnt5a-YAP/TAZ signaling contributes to lung dysfunction in acute exposure to BaP.	Benzo(a)pyrene	209-212	wingless-type MMTV integration site family, member 5A	Mus musculus	132-137