PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28344324-9	2017	We showed that treatment of BGC-823 cells with DATS in vitro and in vivo significantly activated kinases such as p38 and JNK/MAPK and attenuated the Nrf2/Akt pathway.	diallyl trisulfide	47-51	mitogen-activated protein kinase 14	Homo sapiens	113-116	
28344324-0	2017	Diallyl trisulfide suppresses tumor growth through the attenuation of Nrf2/Akt and activation of p38/JNK and potentiates cisplatin efficacy in gastric cancer treatment.	diallyl trisulfide	0-18	mitogen-activated protein kinase 14	Homo sapiens	97-100	
20802973-3	2010	In this study, we investigated the function of p38 mitogen-activated protein kinase (MAPK) and caspase-8 in DATS-induced apoptosis of human CNE2 cells using MTT [3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazolium bromide], flow cytometry assay, and Western blotting.	diallyl trisulfide	108-112	mitogen-activated protein kinase 14	Homo sapiens	47-83	
28501767-6	2017	Besides, activation of JNK, ERK and p38 phosphorylation in DATS-treated cells suggested that mitogen-activated protein kinase (MAPKs) pathways were involved in DATS-induced apoptosis.	diallyl trisulfide	59-63	mitogen-activated protein kinase 14	Homo sapiens	36-39	
25403643-3	2015	In HT29 cells, DATS inhibited migration and invasion through the inhibition of focal adhesion kinase (FAK), extracellular signal-regulated kinase, c-Jun N-terminal kinase and p38 which was associated with inhibition of matrix metalloproteinases-2, -7 and -9 and VEGF.	diallyl trisulfide	15-19	mitogen-activated protein kinase 14	Homo sapiens	175-178