PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26548347-6	2015	The mechanistic study showed that the DATS-mediated inhibition of naphthalene-induced oxidative injury and the production of inflammatory responses (i.e., TNF-alpha, IL-6, and IL-8) were attributed to inhibiting the activity of nuclear factor-kappa B (NF-kappaB).	diallyl trisulfide	38-42	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	228-250	
26548347-6	2015	The mechanistic study showed that the DATS-mediated inhibition of naphthalene-induced oxidative injury and the production of inflammatory responses (i.e., TNF-alpha, IL-6, and IL-8) were attributed to inhibiting the activity of nuclear factor-kappa B (NF-kappaB).	diallyl trisulfide	38-42	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	252-261	
23811270-0	2013	Diallyl trisulfide suppresses dextran sodium sulfate-induced mouse colitis: NF-kappaB and STAT3 as potential targets.	diallyl trisulfide	0-18	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	76-85	
23811270-6	2013	In conclusion, DATS ameliorates the DSS-induced mouse colitis presumably by blocking inflammatory signaling mediated by NF-kappaB and STAT3.	diallyl trisulfide	15-19	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	120-129	
17448272-12	2007	CONCLUSION: Pretreatment of DATS for ALI in mice could inhibit NF-KappaB activity, TNF-alpha mRNA expression in lung tissues, and decrease the release of TNF-alpha in the serum and the lung homogenates, and they might be the underlying mechanisms of prevention of the occurrence of ALI by DATS.	diallyl trisulfide	28-32	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	63-72