PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30301827-9	2019	Mechanistically, MZF1 was upregulated in both GC and GES-1 cells by MT2A ectopically expressed or induced upon treatment with a garlic-derived compound, diallyl trisulfide (DATS).	diallyl trisulfide	153-171	metallothionein 2A	Homo sapiens	68-72	
26801633-0	2016	Epigenetic Upregulation of Metallothionein 2A by Diallyl Trisulfide Enhances Chemosensitivity of Human Gastric Cancer Cells to Docetaxel Through Attenuating NF-kappaB Activation.	diallyl trisulfide	49-67	metallothionein 2A	Homo sapiens	27-45	
26801633-3	2016	The present study investigated the effect of diallyl trisulfide (DATS), a garlic-derived compound, and docetaxel (DOC) on regulation of MT2A in relation to NF-kappaB in GC cells.	diallyl trisulfide	45-63	metallothionein 2A	Homo sapiens	136-140	
26801633-3	2016	The present study investigated the effect of diallyl trisulfide (DATS), a garlic-derived compound, and docetaxel (DOC) on regulation of MT2A in relation to NF-kappaB in GC cells.	diallyl trisulfide	65-69	metallothionein 2A	Homo sapiens	136-140	
26801633-5	2016	The anti-GC effect of DATS was attributable to its capacity to epigenetically upregulate MT2A, which in turn enhanced transcription of IkappaB-alpha to suppress NF-kappaB activation in GC cells.	diallyl trisulfide	22-26	metallothionein 2A	Homo sapiens	89-93	
26801633-9	2016	INNOVATION AND CONCLUSION: We conclude that DATS exerts its anti-GC activity and enhances chemosensitivity of GC to DOC by epigenetic upregulation of MT2A to attenuate NF-kappaB signaling.	diallyl trisulfide	44-48	metallothionein 2A	Homo sapiens	150-154