PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23453443-0	2013	Antioxidant effects of diallyl trisulfide on high glucose-induced apoptosis are mediated by the PI3K/Akt-dependent activation of Nrf2 in cardiomyocytes.	diallyl trisulfide	23-41	AKT serine/threonine kinase 1	Rattus norvegicus	101-104	
28810199-12	2017	In summary, we demonstrated that benzene-induced cytopenia was related to the apoptosis of PBMCs and BMCs, and DATS treatment could prevent benzene-induced cytopenia by suppressing oxidative stress-mediated cell apoptosis via the PI3K/Akt pathway.	diallyl trisulfide	111-115	AKT serine/threonine kinase 1	Rattus norvegicus	235-238	
23453443-8	2013	Inhibition of PI3K/Akt signaling by LY294002 (PI3K inhibitor) or PI3K-specific siRNA not only decreased the level of DATS-induced Nrf2-mediated HO-1 expression, but also diminished the protective effects of DATS.	diallyl trisulfide	117-121	AKT serine/threonine kinase 1	Rattus norvegicus	19-22	
23453443-8	2013	Inhibition of PI3K/Akt signaling by LY294002 (PI3K inhibitor) or PI3K-specific siRNA not only decreased the level of DATS-induced Nrf2-mediated HO-1 expression, but also diminished the protective effects of DATS.	diallyl trisulfide	207-211	AKT serine/threonine kinase 1	Rattus norvegicus	19-22	
23453443-10	2013	CONCLUSIONS: Our findings indicate that DATS protects against hyperglycemia-induced ROS-mediated apoptosis by upregulating the PI3K/Akt/Nrf2 pathway, which further activates Nrf2-regulated antioxidant enzymes in cardiomyocytes exposed to HG.	diallyl trisulfide	40-44	AKT serine/threonine kinase 1	Rattus norvegicus	132-135