PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
29594332-11	2018	Moreover, DATS suppressed the activity of Wnt/beta-catenin pathway, while upregulation of Wnt/beta-catenin diminished the inhibitory effect of DATS on colorectal CSCs.	diallyl trisulfide	10-14	catenin beta 1	Homo sapiens	46-58	
29594332-0	2018	Wnt/beta-catenin signaling mediates the suppressive effects of diallyl trisulfide on colorectal cancer stem cells.	diallyl trisulfide	63-81	catenin beta 1	Homo sapiens	4-16	
29713750-0	2018	Correction to: Wnt/beta-catenin signaling mediates the suppressive effects of diallyl trisulfide on colorectal cancer stem cells.	diallyl trisulfide	78-96	catenin beta 1	Homo sapiens	19-31	
29594332-12	2018	CONCLUSIONS: Wnt/beta-catenin pathway mediates DATS-induced colorectal CSCs suppression.	diallyl trisulfide	47-51	catenin beta 1	Homo sapiens	17-29	
28815294-0	2017	Diallyl trisulfide inhibits proliferation, invasion and angiogenesis of glioma cells by inactivating Wnt/beta-catenin signaling.	diallyl trisulfide	0-18	catenin beta 1	Homo sapiens	105-117	
28815294-7	2017	Consistent with the previous results, diallyl trisulfide inhibited proliferation, invasion and angiogenesis in glioma cells by suppressing Wnt/beta-catenin signaling.	diallyl trisulfide	38-56	catenin beta 1	Homo sapiens	143-155