PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
19622577-0	2009	Transcriptional repression and inhibition of nuclear translocation of androgen receptor by diallyl trisulfide in human prostate cancer cells.	diallyl trisulfide	91-109	androgen receptor	Homo sapiens	70-87	
19622577-1	2009	PURPOSE: The present study was undertaken to determine the effect of diallyl trisulfide (DATS), a promising cancer chemopreventive constituent of garlic, on androgen receptor (AR) protein expression and function using prostate cancer cells.	diallyl trisulfide	69-87	androgen receptor	Homo sapiens	157-174	
19622577-1	2009	PURPOSE: The present study was undertaken to determine the effect of diallyl trisulfide (DATS), a promising cancer chemopreventive constituent of garlic, on androgen receptor (AR) protein expression and function using prostate cancer cells.	diallyl trisulfide	89-93	androgen receptor	Homo sapiens	157-174	
19622577-3	2009	The effect of DATS treatment on AR mRNA level and AR promoter activity was determined by quantitative reverse transcription-PCR and luciferase reporter assay, respectively.	diallyl trisulfide	14-18	androgen receptor	Homo sapiens	32-34	
19622577-3	2009	The effect of DATS treatment on AR mRNA level and AR promoter activity was determined by quantitative reverse transcription-PCR and luciferase reporter assay, respectively.	diallyl trisulfide	14-18	androgen receptor	Homo sapiens	50-52	
19622577-7	2009	RESULTS: Exposure of prostate cancer cells (LNCaP, C4-2, and TRAMP-C1) to DATS resulted in a concentration-dependent decrease in protein level of AR, which was accompanied by suppression of intracellular and secreted levels of PSA.	diallyl trisulfide	74-78	androgen receptor	Homo sapiens	146-148	
19622577-8	2009	Structure-activity studies revealed critical roles for allyl groups and the oligosulfide chain length in DATS-mediated down-modulation of AR protein.	diallyl trisulfide	105-109	androgen receptor	Homo sapiens	138-140	
19622577-12	2009	CONCLUSION: The present study shows, for the first time, that DATS treatment suppresses AR function in prostate cancer cells.	diallyl trisulfide	62-66	androgen receptor	Homo sapiens	88-90