PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
8948524-6	1996	Following gentamicin treatment, increased NAG, AAP, and GGT enzymuria were noted at all time points tested.	Gentamicins	10-20	N-acetyl-alpha-glucosaminidase	Homo sapiens	42-45	
9279185-4	1997	Increased NAG values reflect the degree of perinatal asphyxia more than do beta 2 M. Gentamicin also increased NAG excretion, but to a lesser extent than did perinatal asphyxia.	Gentamicins	85-95	N-acetyl-alpha-glucosaminidase	Homo sapiens	111-114	
3283687-0	1988	[Urinary excretion of N-acetyl-beta-D-glucosaminidase (NAG) and its isoenzyme B as a marker of the nephrotoxicity of gentamicin: re-test from an animal model].	Gentamicins	117-127	N-acetyl-alpha-glucosaminidase	Homo sapiens	55-58	
7487376-6	1994	The use of two types of potentially nephrotoxic drugs, like NSAID and gentamicin or NSAID with cyclosporin induced significant augmentation of NAG excretion.	Gentamicins	70-80	N-acetyl-alpha-glucosaminidase	Homo sapiens	143-146	
3283687-2	1988	The urinary excretion of NAG following gentamicin treatment was studied in rabbit.	Gentamicins	39-49	N-acetyl-alpha-glucosaminidase	Homo sapiens	25-28	
3283687-8	1988	Surprisingly, the high level of NAG excretion following the administration of gentamicin at 50 mg/kg/j is not accompanied by a marked excretion of isoenzyme B.	Gentamicins	78-88	N-acetyl-alpha-glucosaminidase	Homo sapiens	32-35	
3283687-10	1988	These data indicate that increases in urinary NAG and isoenzyme B excretion following gentamicin treatment in Human are not always reliable indicators of renal tubular cell lysis.	Gentamicins	86-96	N-acetyl-alpha-glucosaminidase	Homo sapiens	46-49