PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 8948524-6 1996 Following gentamicin treatment, increased NAG, AAP, and GGT enzymuria were noted at all time points tested. Gentamicins 10-20 N-acetyl-alpha-glucosaminidase Homo sapiens 42-45 9279185-4 1997 Increased NAG values reflect the degree of perinatal asphyxia more than do beta 2 M. Gentamicin also increased NAG excretion, but to a lesser extent than did perinatal asphyxia. Gentamicins 85-95 N-acetyl-alpha-glucosaminidase Homo sapiens 111-114 7487376-6 1994 The use of two types of potentially nephrotoxic drugs, like NSAID and gentamicin or NSAID with cyclosporin induced significant augmentation of NAG excretion. Gentamicins 70-80 N-acetyl-alpha-glucosaminidase Homo sapiens 143-146 3283687-0 1988 [Urinary excretion of N-acetyl-beta-D-glucosaminidase (NAG) and its isoenzyme B as a marker of the nephrotoxicity of gentamicin: re-test from an animal model]. Gentamicins 117-127 N-acetyl-alpha-glucosaminidase Homo sapiens 55-58 3283687-2 1988 The urinary excretion of NAG following gentamicin treatment was studied in rabbit. Gentamicins 39-49 N-acetyl-alpha-glucosaminidase Homo sapiens 25-28 3283687-8 1988 Surprisingly, the high level of NAG excretion following the administration of gentamicin at 50 mg/kg/j is not accompanied by a marked excretion of isoenzyme B. Gentamicins 78-88 N-acetyl-alpha-glucosaminidase Homo sapiens 32-35 3283687-10 1988 These data indicate that increases in urinary NAG and isoenzyme B excretion following gentamicin treatment in Human are not always reliable indicators of renal tubular cell lysis. Gentamicins 86-96 N-acetyl-alpha-glucosaminidase Homo sapiens 46-49