PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
10772385-2	2000	In order to confirm and expand these studies, we have compared several H1-blockers and the H2-blocker ranitidine for their effect on TNF-alpha, IL-3, 6, 8 and GM-CSF release from human leukemic mast (HMC-1) and basophilic (KU812) cells, compared to dexamethasone.	Ranitidine	102-112	tumor necrosis factor	Homo sapiens	133-142	
10772385-4	2000	All antihistamines caused a dose-dependent inhibition of TNF-alpha release from HMC-1 cells, with maximal effects at 10(-12) M for azelastine, 10(-9) M for loratadine and cetirizine, and 10(-8) M for ranitidine.	Ranitidine	200-210	tumor necrosis factor	Homo sapiens	57-66	
10772385-7	2000	Dexamethasone inhibited all cytokines, but ranitidine only TNF-alpha and IL-3.	Ranitidine	43-53	tumor necrosis factor	Homo sapiens	59-68	
7809839-12	1995	Ranitidine delayed the LPS-induced ventilation-perfusion imbalance and attenuated the peak increase in the proinflammatory cytokine, tumor necrosis factor, without altering its antiinflammatory opponent, cortisol.	Ranitidine	0-10	tumor necrosis factor	Homo sapiens	133-154	
2056280-6	1991	The histamine-mediated decrease in TNF-alpha synthesis was not affected by indomethacin, nor by diphenhydramine, an H1 receptor antagonist, but was reversed by cimetidine or ranitidine, H2 receptor antagonists, in a dose-dependent manner.	Ranitidine	174-184	tumor necrosis factor	Homo sapiens	35-44	
9602986-0	1998	Changes in mucosal levels of transforming growth factor-alpha from the oxyntic region and ulcer site during duodenal ulcer healing with ranitidine or sucralfate.	Ranitidine	136-146	tumor necrosis factor	Homo sapiens	29-61