PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 14557432-13 2003 The fall in CRP and IL-6 was more pronounced with the high- than low-dose vitamin D (P < 0.05). Vitamin D 74-83 C-reactive protein Homo sapiens 12-15 15780504-5 2005 In a recent clinical trial targeting elderly chronically ill patients, administration of vitamin D reduced serum levels of both CRP and IL-6; further such studies should assess the impact of physiologically meaningful doses of vitamin D on acute phase reactants in elderly subjects likely to have poor vitamin D status. Vitamin D 89-98 C-reactive protein Homo sapiens 128-131 12454321-9 2002 RESULTS: Vitamin D status was the sole determinant of circulating MMP9 (inversely) and an independent determinant of CRP (inversely). Vitamin D 9-18 C-reactive protein Homo sapiens 117-120 12454321-12 2002 DISCUSSION: Vitamin-D insufficiency is associated with increased circulating MMP2,9 and CRP, correctable by supplementation. Vitamin D 12-21 C-reactive protein Homo sapiens 77-91 33807159-2 2021 We examined the cross-sectional association between biomarkers of vitamin D status and C-reactive protein (CRP) among postmenopausal women aged 53-81 years. Vitamin D 66-75 C-reactive protein Homo sapiens 87-105 33818731-7 2021 Higher C-reactive protein (CRP) levels were seen in the vitamin D-deficient group (p = 0.013). Vitamin D 56-65 C-reactive protein Homo sapiens 7-25 33818731-7 2021 Higher C-reactive protein (CRP) levels were seen in the vitamin D-deficient group (p = 0.013). Vitamin D 56-65 C-reactive protein Homo sapiens 27-30 33807159-2 2021 We examined the cross-sectional association between biomarkers of vitamin D status and C-reactive protein (CRP) among postmenopausal women aged 53-81 years. Vitamin D 66-75 C-reactive protein Homo sapiens 107-110 34839468-6 2021 In the study group, the neonatal 25-OH vitamin D was negatively correlated with C-reactive protein and length of hospital stay (r = -0.616 and -0.596, respectively) p <0.001 for both. Vitamin D 39-48 C-reactive protein Homo sapiens 80-98 34944509-11 2021 High doses of vitamin D supplementation led to a significant decrease in pro-inflammatory cytokines (IFN- , TNF-alpha, IL-1beta, IL-6, IL-8, and IL-17) and high-sensitivity C-reactive protein (hsCRP), whereas the production of anti-inflammatory cytokines (IL-10, IL-5) was up-regulated. Vitamin D 14-23 C-reactive protein Homo sapiens 173-191 34857202-7 2021 RESULTS: There was a noticeable regular trend regarding hypercholesterolemia (p = 0.008), high LDL-C (p = 0.024), hypertension (p = 0.021), and high hs-CRP (p < 0.0001) across various categories of vitamin D status. Vitamin D 198-207 C-reactive protein Homo sapiens 152-155 34857202-8 2021 In adjusted model, vitamin D-deficient subjects were at higher risk for having hypercholesterolemia (OR: 3.22, p = 0.031), high LDL-C (OR: 2.37, p = 0.047), hypertension (OR: 2.32, p = 0.042), and high hs-CRP (OR: 5.49, p = 0.001) than ones with sufficient vitamin D status. Vitamin D 19-28 C-reactive protein Homo sapiens 205-208 34857202-9 2021 CONCLUSIONS: Vitamin D deficiency in obese subjects was found to be strongly related to higher risk of unfavorable lipid profile, hypertension, and high hs-CRP. Vitamin D 13-22 C-reactive protein Homo sapiens 156-159 34840966-7 2021 Vitamin D showed a significant negative correlation with LDH, CRP, ESR, ferritin, and D-dimer, which was the most reliable predictor of COVID-19 severity in T1DM patients. Vitamin D 0-9 C-reactive protein Homo sapiens 62-65 34857984-8 2021 The positive effects of vitamin D supplementation on body mass index, waist, HDL-C, LDL-C, and CRP were also demonstrated. Vitamin D 24-33 C-reactive protein Homo sapiens 95-98 34781136-9 2021 PRACTICAL APPLICATION: The findings of the present study indicated that a low-fat yogurt fortified with 1500 IU nano-encapsulated vitamin D for ten weeks, leads to a significant reduction in serum hs-CRP and PAB concentrations highlighted the anti-inflammatory/anti-oxidative effect of vitamin D. Vitamin D 130-139 C-reactive protein Homo sapiens 200-203 34781136-9 2021 PRACTICAL APPLICATION: The findings of the present study indicated that a low-fat yogurt fortified with 1500 IU nano-encapsulated vitamin D for ten weeks, leads to a significant reduction in serum hs-CRP and PAB concentrations highlighted the anti-inflammatory/anti-oxidative effect of vitamin D. Vitamin D 286-295 C-reactive protein Homo sapiens 200-203 34375576-9 2021 C-reactive protein level was higher in the low vitamin D group, although the difference did not reach statistical significance (9.6 +- 2.2 vs. 4.5 +- 1.6 mg/l, P = 0.074). Vitamin D 47-56 C-reactive protein Homo sapiens 0-18 34174792-8 2021 CRP,TNF-?, IL-6 and IL-10 levels were also correlated with serum vitamin D levels (p <0.05). Vitamin D 65-74 C-reactive protein Homo sapiens 0-9 34744508-10 2021 Vitamin D level had negative correlations with ferritin (R=-0.316, p=0.044) and CRP (R=-0.530, p=0.000). Vitamin D 0-9 C-reactive protein Homo sapiens 80-83 34464543-11 2021 COVID-19 patients with low vitamin D levels had a greater prevalence of hypertension and cardiovascular diseases, abnormally high serum troponin and peak D-dimer levels, as well as elevated interleukin-6 and C-reactive protein than those with serum 25(OH)D levels >=30 ng/ml. Vitamin D 27-36 C-reactive protein Homo sapiens 208-226 34267439-6 2021 A significant inverse correlation was found between vitamin D level and 24 h protein in urine, ANA, anti-dsDNA, CRP, with a significant positive correlation with renal biopsy indices, eGFR. Vitamin D 52-61 C-reactive protein Homo sapiens 112-115 34126229-2 2021 Therefore, the present study was conducted on AGH patients to assess the impact of vitamin D on inflammatory cytokines such as CRP, TNF-alpha and IL-6. Vitamin D 83-92 C-reactive protein Homo sapiens 127-130 34126229-10 2021 Summarizing earlier studies, we demonstrated that circulating concentrations of inflammatory cytokines such as CRP, TNF-alpha, and IL-6 might be decreased following vitamin D supplementation among individuals with AGH. Vitamin D 165-174 C-reactive protein Homo sapiens 111-114 34347528-8 2021 Lower vitamin D levels were significantly associated with more severe COVID-19 cases (p-value < 0.001), higher blood levels of inflammatory markers including (D-dimer, CRP, and ferritin), a higher CT SS and longer disease duration. Vitamin D 6-15 C-reactive protein Homo sapiens 168-171 35631138-1 2022 The aim of the study was to evaluate the vitamin D status in hospitalized COVID-19 patients and the correlation with C reactive protein (CRP), ferritin, fibrinogen, and peripheral blood leukocytes, as well as inflammatory derived indices. Vitamin D 41-50 C-reactive protein Homo sapiens 137-140 35579027-1 2022 BACKGROUND: Low vitamin D status is often associated with systemic low-grade inflammation as reflected by elevated C-reactive protein (CRP) levels. Vitamin D 16-25 C-reactive protein Homo sapiens 115-133 35579027-1 2022 BACKGROUND: Low vitamin D status is often associated with systemic low-grade inflammation as reflected by elevated C-reactive protein (CRP) levels. Vitamin D 16-25 C-reactive protein Homo sapiens 135-138 35579027-2 2022 We investigated the causality and direction of the association between vitamin D status and CRP using linear and non-linear Mendelian randomization (MR) analyses. Vitamin D 71-80 C-reactive protein Homo sapiens 92-95 35579027-8 2022 CONCLUSION: The observed association between 25(OH)D and CRP is likely to be caused by vitamin D deficiency. Vitamin D 87-96 C-reactive protein Homo sapiens 57-60 35535979-10 2022 In participants with high-carbohydrate/low-fat diets and low vitamin D intakes, those with High-PRS had a higher risk of serum CRP concentrations than those with Low-PRS. Vitamin D 61-70 C-reactive protein Homo sapiens 127-130 33974699-0 2021 Retraction of Vitamin D Supplementation Affects Serum High-Sensitivity C-Reactive Protein, Insulin Resistance, and Biomarkers of Oxidative Stress in Pregnant Women. Vitamin D 14-23 C-reactive protein Homo sapiens 71-89 35415272-11 2022 Serum concentrations of vitamin D were positively correlated with levels of serum calcium, lymphocytes, and neutrophils but negatively correlated with CRP, fibrinogen, and d-dimer values. Vitamin D 24-33 C-reactive protein Homo sapiens 151-154 35443576-9 2022 Moreover, vitamin D levels showed significant negative correlation with the inflammatory markers: TNF-alpha, CRP, and HIF-1alpha levels. Vitamin D 10-19 C-reactive protein Homo sapiens 109-112 35277211-8 2022 There was a negative correlation between vitamin D status and CRP levels (=-.364, p=.007). Vitamin D 41-50 C-reactive protein Homo sapiens 62-65 35228490-12 2022 The serum C-reactive protein showed significant reduction (p=0.028*) after intervention with vitamin D. Vitamin D 93-102 C-reactive protein Homo sapiens 10-28 35228490-14 2022 It was concluded that a single large dose of vitamin D was able to reduce the C-reactive protein in non-ST-elevation acute coronary syndrome patients while non-significant reductions in interleukin-6 and tumor necrosis factor-alpha were observed. Vitamin D 45-54 C-reactive protein Homo sapiens 78-96 34033278-0 2021 Moderator role of vitamin D concentrations on the association between metabolic syndrome and C-reactive protein among adults. Vitamin D 18-27 C-reactive protein Homo sapiens 93-111 35458206-5 2022 Vitamin D deficiency was found in 67.2% and was independent of sex, disease manifestation, and CRP, ESR, ALP, or PO4 levels. Vitamin D 0-9 C-reactive protein Homo sapiens 95-98 34989171-10 2022 In addition, the serum inflammation factors (CRP, IL6, and TNF-alpha) were significantly reduced by vitamin D supplementation. Vitamin D 100-109 C-reactive protein Homo sapiens 45-48 35057447-11 2022 In individuals with chronic pain, based on clinical categories, significant associations between vitamin D, omega 6:3 ratio, and CRP were observed. Vitamin D 97-106 C-reactive protein Homo sapiens 129-132 35576543-1 2022 OBJECTIVES: This study is aimed to determine the relationship between 25-OH vitamin D levels, inflammatory parameters of neutrophil-to-lymphocyte ratio (NLR), platelet-to-lymphocyte ratio (PLR), c-reactive protein (CRP) levels and the disease severity of COVID-19 infection. Vitamin D 76-85 C-reactive protein Homo sapiens 195-213 35576543-1 2022 OBJECTIVES: This study is aimed to determine the relationship between 25-OH vitamin D levels, inflammatory parameters of neutrophil-to-lymphocyte ratio (NLR), platelet-to-lymphocyte ratio (PLR), c-reactive protein (CRP) levels and the disease severity of COVID-19 infection. Vitamin D 76-85 C-reactive protein Homo sapiens 215-218 35379386-10 2022 Meta-analysis showed that patients that were vitamin D sufficient (levels >30ng/mL) had statistically significant lower levels of IL-6, CRP, ferritin, LDH, fibrinogen, and D-dimer compared to vitamin D deficient group. Vitamin D 45-54 C-reactive protein Homo sapiens 136-139 33788001-8 2021 The 25 OH vitamin D level was correlated positively with the lymphocyte count (r = 0.375, p = <0.001), and negatively with age (r = -0.496, p = <0.001), CRP (r = -0.309, p = 0.002) and fibrinogen levels (r = -0.381, p = <0.001). Vitamin D 10-19 C-reactive protein Homo sapiens 153-156 33512007-5 2021 CONCLUSION: Elevated vitamin D levels could decrease COVID-19 PCR positivity,D-dimer and CRP levels and the number of affected lung segments in COVID-19 positive patients,thereby shortening duration of hospital stays and alleviating the intensity of COVID-19. Vitamin D 21-30 C-reactive protein Homo sapiens 89-92 33112413-9 2021 The levels of white blood cells, leukocytes subfamilies, and inflammatory parameters significantly correlated with vitamin D levels in both patients with and without diabetes (diabetic: p=0.012 for WBC, p=0.004 for NLR and p<0.001 for MLR and C-reactive protein, non-diabetic: p<0.001 for WBC; NLR, MLR and C reactive protein, respectively). Vitamin D 115-124 C-reactive protein Homo sapiens 243-261 33112413-9 2021 The levels of white blood cells, leukocytes subfamilies, and inflammatory parameters significantly correlated with vitamin D levels in both patients with and without diabetes (diabetic: p=0.012 for WBC, p=0.004 for NLR and p<0.001 for MLR and C-reactive protein, non-diabetic: p<0.001 for WBC; NLR, MLR and C reactive protein, respectively). Vitamin D 115-124 C-reactive protein Homo sapiens 307-325 33454206-8 2021 There was a negative correlation between vitamin D status and CRP levels (=-.364, p=.007). Vitamin D 41-50 C-reactive protein Homo sapiens 62-65 33677945-9 2021 25-OH-vitamin D deficiency was associated with increased CRP and dyspnea. Vitamin D 6-15 C-reactive protein Homo sapiens 57-60 33726658-14 2021 A significant negative correlation found between vitamin D and ALT (P= 0.02, -0.21) as well as vitamin D and CRP (P= 0.05, -0.17). Vitamin D 95-104 C-reactive protein Homo sapiens 109-112 33672176-5 2021 In all subjects, vitamin D was negatively associated with c-reactive protein (CRP) (p < 0.001) and with probing depth (PD), clinical attachment level (CAL), bleeding on probing (BOP), and plaque score (PI) (p < 0.001 for all parameters) and positively related to the number of teeth (p < 0.001). Vitamin D 17-26 C-reactive protein Homo sapiens 58-76 33672176-5 2021 In all subjects, vitamin D was negatively associated with c-reactive protein (CRP) (p < 0.001) and with probing depth (PD), clinical attachment level (CAL), bleeding on probing (BOP), and plaque score (PI) (p < 0.001 for all parameters) and positively related to the number of teeth (p < 0.001). Vitamin D 17-26 C-reactive protein Homo sapiens 78-81 33672176-7 2021 The multivariate regression analysis showed that PT (p = 0.011) and CRP (p = 0.031) were both predictors of vitamin D levels. Vitamin D 108-117 C-reactive protein Homo sapiens 68-71 33341448-5 2021 RESULTS: Across all tumour stages, vitamin D-deficient patients had the highest median levels of IL-6 (8.3 pg/mL, range 0.7-91), YKL-40 (177 ng/ml, range 25-5279) and CRP (15.5 mg/L, range 0.8-384). Vitamin D 35-44 C-reactive protein Homo sapiens 167-170 33508990-6 2021 Moreover, vitamin D intake led to a significant reduction in high-sensitivity C-reactive protein (hs-CRP) (beta - 0.64 mg/L; 95% CI, -0.97, -0.30; p<.001) and a significant increase in total antioxidant capacity (TAC) (beta 47.54 mmol/L; 95% CI, 19.98, 75.11; p=.001) compared with the placebo. Vitamin D 10-19 C-reactive protein Homo sapiens 78-96 33508990-7 2021 CONCLUSIONS: Overall, our study demonstrated that vitamin D intake in patients with endometriosis resulted in a significant improvement of pelvic pain, total-/HDL-cholesterol ratio, hs-CRP and TAC levels, but did not affect other clinical symptoms and metabolic profiles. Vitamin D 50-59 C-reactive protein Homo sapiens 185-188 33545754-8 2021 The pooled results demonstrated that vitamin D supplementation in patients with PCOS resulted in a significant improvement in serum total testosterone (TT), high sensitivity C-reactive protein (hs-CRP), total antioxidant capacity (TAC), and malondialdehyde (MDA). Vitamin D 37-46 C-reactive protein Homo sapiens 174-192 33545754-8 2021 The pooled results demonstrated that vitamin D supplementation in patients with PCOS resulted in a significant improvement in serum total testosterone (TT), high sensitivity C-reactive protein (hs-CRP), total antioxidant capacity (TAC), and malondialdehyde (MDA). Vitamin D 37-46 C-reactive protein Homo sapiens 197-200 33545754-10 2021 Subgroup analysis showed that vitamin D supplementation reduced hs-CRP and MDA irrespective of the treatment course, type of vitamin D intervention, supplementation frequency, and dosage. Vitamin D 30-39 C-reactive protein Homo sapiens 67-70 33545754-12 2021 CONCLUSIONS: The current meta-analysis demonstrates that vitamin D supplementation in patients with PCOS resulted in an improvement in the levels of TT, hs-CRP, TAC, and MDA, but did not affect FT, DHEA-S, SHBG, FAI, NO, and GSH levels. Vitamin D 57-66 C-reactive protein Homo sapiens 156-159 32422661-6 2020 We found that vitamin D supplementation had a significant effect on insulin metabolism, total serum testosterone, hirsutism, C-reactive protein, and total antioxidant capacity in women with polycystic ovary syndrome. Vitamin D 14-23 C-reactive protein Homo sapiens 125-143 33097953-0 2020 An Expression of Concern from The Journal of Nutrition"s Editorial Office about: Vitamin D Supplementation Affects Serum High-Sensitivity C-Reactive Protein, Insulin Resistance, and Biomarkers of Oxidative Stress in Pregnant Women. Vitamin D 81-90 C-reactive protein Homo sapiens 138-156 32408752-9 2020 In multivariate analysis, only AIDS status and CRP level were correlated with vitamin D level (slope estimate = 11.6 and p = 0.032 and slope estimate = -0.83 and p = 0.002; respectively). Vitamin D 78-87 C-reactive protein Homo sapiens 47-50 32408752-10 2020 In summary, we report that low vitamin D level may be associated with high CRP level in HIV-infected patients on suppressive antiretroviral therapy, especially in AIDS phase. Vitamin D 31-40 C-reactive protein Homo sapiens 75-78 32684941-0 2020 Vitamin D status affects the relationship between lipid profile and high-sensitivity C-reactive protein. Vitamin D 0-9 C-reactive protein Homo sapiens 85-103 32684941-2 2020 Objective: The objective of this study was to evaluate the role of vitamin D status on the relationship between lipid profile and high-sensitivity C-reactive protein (hs-CRP) in pregnant women. Vitamin D 67-76 C-reactive protein Homo sapiens 147-165 32684941-2 2020 Objective: The objective of this study was to evaluate the role of vitamin D status on the relationship between lipid profile and high-sensitivity C-reactive protein (hs-CRP) in pregnant women. Vitamin D 67-76 C-reactive protein Homo sapiens 170-173 32684941-11 2020 Conclusion: Our findings suggest that high levels of vitamin D during pregnancy may improve lipid profile levels and inhibit elevated hs-CRP induced by high lipid metabolism. Vitamin D 53-62 C-reactive protein Homo sapiens 137-140 32659891-6 2020 RESULTS: The vitamin D group showed higher blood levels of (25(OH) D) (p < 0.0001), and a significant reduction in hs-CRP and TNF-alpha concentrations (p < 0.0001) compared to placebo. Vitamin D 13-22 C-reactive protein Homo sapiens 118-121 32325367-0 2020 Adiposity is a confounding factor which largely explains the association of serum vitamin D concentrations with C-reactive protein, leptin and adiponectin. Vitamin D 82-91 C-reactive protein Homo sapiens 112-130 33224249-5 2020 Results: Vitamin D supplementation after the intervention led to a significant decrease in triglycerides (TG) (P = 0.02), very-low-density lipoprotein-cholesterol (VLDL-C) (P = 0.02), and hs-CRP (P = 0.03) concentrations and a significant increase in the serum vitamin D level (P < 0.001). Vitamin D 9-18 C-reactive protein Homo sapiens 191-194 32976513-4 2020 After adjusting for confounding factors, there was a significant association between vitamin D sufficiency and reduction in clinical severity, inpatient mortality serum levels of C-reactive protein (CRP) and an increase in lymphocyte percentage. Vitamin D 85-94 C-reactive protein Homo sapiens 179-197 32976513-4 2020 After adjusting for confounding factors, there was a significant association between vitamin D sufficiency and reduction in clinical severity, inpatient mortality serum levels of C-reactive protein (CRP) and an increase in lymphocyte percentage. Vitamin D 85-94 C-reactive protein Homo sapiens 199-202 32976513-6 2020 The significant reduction in serum CRP, an inflammatory marker, along with increased lymphocytes percentage suggest that vitamin D sufficiency also may help modulate the immune response possibly by reducing risk for cytokine storm in response to this viral infection. Vitamin D 121-130 C-reactive protein Homo sapiens 35-38 32968400-8 2020 The mechanism may be associated with the impact of vitamin D deficiency on hs-CRP and other body inflammation indicators, as well as on thyroid autoantibodies and other immune indicators, but has no effect on IL-1 levels. Vitamin D 51-60 C-reactive protein Homo sapiens 78-81 32408752-6 2020 Furthermore, the significant negative correlation between vitamin D level and CD8+ cell percentage, PLT, CRP and D-dimers was seen. Vitamin D 58-67 C-reactive protein Homo sapiens 105-108 32884931-3 2020 Objectives: This study aims at investigating the relationship between vitamin D, fasting plasma glucose (FPG), insulin, zinc, ferritin, and high-sensitivity C-reactive protein (CRP) in GDM. Vitamin D 70-79 C-reactive protein Homo sapiens 157-175 32884931-3 2020 Objectives: This study aims at investigating the relationship between vitamin D, fasting plasma glucose (FPG), insulin, zinc, ferritin, and high-sensitivity C-reactive protein (CRP) in GDM. Vitamin D 70-79 C-reactive protein Homo sapiens 177-180 32884931-8 2020 In the GDM group, a positive weak relationship was observed between vitamin D and zinc (r = 0.18, p < 0.05), while vitamin D was inversely correlated with FPG, serum insulin, ferritin, and CRP (r = -0.23, -0.21, -0.20, -0.46, respectively, p < 0.05). Vitamin D 115-124 C-reactive protein Homo sapiens 189-192 32099860-8 2020 Vitamin D was positively correlated with CRP, ESR level, the BASFI, and the BASMI. Vitamin D 0-9 C-reactive protein Homo sapiens 41-44 33877102-7 2020 Vitamin D had a significant negative correlation with US-CRP (p = 0.026), NLR (p = 0.013) and PLR (p = 0.022). Vitamin D 0-9 C-reactive protein Homo sapiens 57-60 33877102-9 2020 The values of US-CRP, NLR and PLR were significantly higher in the presence of vitamin D deficiency. Vitamin D 79-88 C-reactive protein Homo sapiens 17-20 33877102-10 2020 A significant inverse correlation was found between vitamin D levels and US-CRP, NLR and PLR. Vitamin D 52-61 C-reactive protein Homo sapiens 76-79 32113022-6 2020 Pooling 9 effect sizes from 8 RCTs on C-reactive protein (CRP) levels revealed a significant reducing effect of vitamin d-calcium co-supplementation on serum CRP concentrations compared to placebo intake (WMD: -0.82, 95% CI: -1.56, -0.07 mg/L, P = 0.03). Vitamin D 112-121 C-reactive protein Homo sapiens 38-56 32113022-6 2020 Pooling 9 effect sizes from 8 RCTs on C-reactive protein (CRP) levels revealed a significant reducing effect of vitamin d-calcium co-supplementation on serum CRP concentrations compared to placebo intake (WMD: -0.82, 95% CI: -1.56, -0.07 mg/L, P = 0.03). Vitamin D 112-121 C-reactive protein Homo sapiens 58-61 32113022-6 2020 Pooling 9 effect sizes from 8 RCTs on C-reactive protein (CRP) levels revealed a significant reducing effect of vitamin d-calcium co-supplementation on serum CRP concentrations compared to placebo intake (WMD: -0.82, 95% CI: -1.56, -0.07 mg/L, P = 0.03). Vitamin D 112-121 C-reactive protein Homo sapiens 158-161 32113022-8 2020 Also, we found that the associations of vitamin d-calcium dosages and duration of intervention with the reduction in CRP concentrations were in a non-linear fashion. Vitamin D 40-49 C-reactive protein Homo sapiens 117-120 32113022-10 2020 We found a beneficial effect of vitamin d-calcium co-supplementation on serum CRP concentrations. Vitamin D 32-41 C-reactive protein Homo sapiens 78-81 32188088-12 2020 In patients with serum vitamin D below 25 ng/mL, vitamin D seasonal change correlated with CRP and prealbumin change. Vitamin D 23-32 C-reactive protein Homo sapiens 91-94 32188088-12 2020 In patients with serum vitamin D below 25 ng/mL, vitamin D seasonal change correlated with CRP and prealbumin change. Vitamin D 49-58 C-reactive protein Homo sapiens 91-94 31255367-1 2020 INTRODUCTION AND OBJECTIVES: Previous observational studies have suggested that low vitamin D status is associated with high circulating C-reactive protein levels, as well as other plasma inflammatory cytokines. Vitamin D 84-93 C-reactive protein Homo sapiens 137-155 32147032-0 2020 The effect of intramuscular megadose of vitamin D injections on E-selectin, CRP and biochemical parameters in vitamin D-deficient patients with type-2 diabetes mellitus: A randomized controlled trial. Vitamin D 40-49 C-reactive protein Homo sapiens 76-79 32147032-6 2020 RESULTS: Vitamin D resulted in significant reductions in CRP(P = 0.01) and gamma glutamyl transferase (GGT) levels(P = 0.03) and significant increases in 25(OH)D concentrations(P = 0.01) in the intervention group compared with the control. Vitamin D 9-18 C-reactive protein Homo sapiens 57-60 32147032-9 2020 CONCLUSION: Vitamin D had beneficial effects on the levels of CRP, serum 25(OH)D and GGT among vitamin D deficient patients with T2DM. Vitamin D 12-21 C-reactive protein Homo sapiens 62-65 31032644-6 2019 Vitamin D supplementation in patients with CHF improved health-related quality of life and C-reactive protein levels [weighted mean difference (WMD): 6.75, 95% confidence interval (CI): 2.87 to 10.64, p < .001; standardised mean difference (SMD): -0.41, 95% CI: -0.71 to -0.11, p = .007]. Vitamin D 0-9 C-reactive protein Homo sapiens 91-109 31731651-7 2019 Vitamin D supplementation was associated with a decreasing trend of iPTH and C-reactive protein (CRP) (p < 0.001). Vitamin D 0-9 C-reactive protein Homo sapiens 77-95 31731651-7 2019 Vitamin D supplementation was associated with a decreasing trend of iPTH and C-reactive protein (CRP) (p < 0.001). Vitamin D 0-9 C-reactive protein Homo sapiens 97-100 31723229-0 2019 The Vitamin D status is associated with serum C-reactive protein and adhesion molecules in patients with renal cell carcinoma. Vitamin D 4-13 C-reactive protein Homo sapiens 46-64 31723229-2 2019 This study investigated the association of vitamin D status with serum C-reactive protein (CRP) and adhesion molecules among RCC patients. Vitamin D 43-52 C-reactive protein Homo sapiens 71-89 31723229-2 2019 This study investigated the association of vitamin D status with serum C-reactive protein (CRP) and adhesion molecules among RCC patients. Vitamin D 43-52 C-reactive protein Homo sapiens 91-94 30885444-0 2019 Vitamin D deficiency in a psychiatric population and correlation between vitamin D and CRP. Vitamin D 73-82 C-reactive protein Homo sapiens 87-90 31338797-0 2019 The effects of vitamin D treatment on glycemic control, serum lipid profiles, and C-reactive protein in patients with chronic kidney disease: a systematic review and meta-analysis of randomized controlled trials. Vitamin D 15-24 C-reactive protein Homo sapiens 82-100 31338797-10 2019 CONCLUSIONS: This meta-analysis demonstrated the beneficial effects of vitamin D supplementation or treatment on improving fasting glucose, HOMA-IR, triglycerides and cholesterol levels among patients with CKD, though it did not influence insulin, HbA1c, LDL and HDL cholesterol, and CRP levels. Vitamin D 71-80 C-reactive protein Homo sapiens 284-287 30097651-10 2019 After adjustment for lipid markers, age, and gender, vitamin D deficiency was associated with increased odds of CRP, eGFR, gammaGT, FPG, HbA1c, and the surrogate for CVD. Vitamin D 53-62 C-reactive protein Homo sapiens 112-115 31356558-8 2019 Increase in vitamin D levels correlated with reduction in UCDAI score (P<=0.001; rho=-0.713), C-reactive protein (P<=0.001; rho=-0.603), and calprotectin (P=0.004; rho=-0.368). Vitamin D 12-21 C-reactive protein Homo sapiens 94-112 30459099-7 2019 Vitamin D and probiotic co-supplementation significantly decreased triglycerides (P = 0.02), VLDL-cholesterol (P = 0.02) and hs-CRP (P = 0.01), and significantly increased TAC (P = 0.006) and total glutathione levels (P = 0.04) compared with only probiotic group. Vitamin D 0-9 C-reactive protein Homo sapiens 128-131 31385179-13 2019 CONCLUSION: CRP levels and NLR values were significantly higher in the vitamin D deficiency group. Vitamin D 71-80 C-reactive protein Homo sapiens 12-15 31385179-14 2019 A significant inverse correlation was found between serum vitamin D levels and CRP levels, and NLR and PLR values. Vitamin D 58-67 C-reactive protein Homo sapiens 79-82 31095994-9 2019 Combining data from five studies, we found a significant reduction in C-reactive protein (CRP) concentrations after vitamin D supplementation (WMD: -1.74; 95% CI: -2.82, -0.66). Vitamin D 116-125 C-reactive protein Homo sapiens 70-88 31095994-9 2019 Combining data from five studies, we found a significant reduction in C-reactive protein (CRP) concentrations after vitamin D supplementation (WMD: -1.74; 95% CI: -2.82, -0.66). Vitamin D 116-125 C-reactive protein Homo sapiens 90-93 31095994-10 2019 CONCLUSIONS: Overall, the current meta-analysis demonstrated that taking vitamin D supplements among patients with psychiatric disorders had beneficial effects on BDI, PSQI, GSH, TAC and CRP levels, but did not affect other biomarkers of inflammation and oxidative stress. Vitamin D 73-82 C-reactive protein Homo sapiens 187-190 31311491-1 2019 BACKGROUND: This study aimed to evaluate the association between serum vitamin D levels and nonalcoholic fatty liver disease (NAFLD) parameters, such as metabolic syndrome (MS), inflammatory cytokines (tumor necrosis factor, high sensitive C-reactive protein) and adipokines (adiponectin, leptin). Vitamin D 71-80 C-reactive protein Homo sapiens 240-258 31020672-8 2019 In stratified analysis, a significant positive association between vitamin D level and CVD was observed only in the high CRP group. Vitamin D 67-76 C-reactive protein Homo sapiens 121-124 31020672-10 2019 CONCLUSION: Within a cross-sectional, nationally representative sample, these findings suggest that vitamin D status evaluation, or vitamin D supplement may be especially important for individuals with high CRP levels. Vitamin D 100-109 C-reactive protein Homo sapiens 207-210 31020672-10 2019 CONCLUSION: Within a cross-sectional, nationally representative sample, these findings suggest that vitamin D status evaluation, or vitamin D supplement may be especially important for individuals with high CRP levels. Vitamin D 132-141 C-reactive protein Homo sapiens 207-210 31176378-8 2019 Skin fold thickness, fasting and post-glucose insulin, HOMA-IR, PTH, LDL, Serum cholesterol and hs-CRP showed statistically significant negative correlations with Vitamin D levels. Vitamin D 163-172 C-reactive protein Homo sapiens 99-102 31023051-9 2019 CONCLUSION: Our results showed that vitamin D deficiency predisposed to the development of sepsis, negatively correlated with CRP, presepsin, sTREM-1 and SOFA score and their levels associates with both 7th and 28th days survival of patients (Tab. Vitamin D 36-45 C-reactive protein Homo sapiens 126-129 29752008-13 2019 Interestingly, the Hs-CRP was reduced in AA carries while was elevated in individuals with GG and AG genotypes, after high-dose vitamin D supplementation. Vitamin D 128-137 C-reactive protein Homo sapiens 22-25 30477283-5 2019 RESULTS: In 88 patients with Crohn"s disease (CD), a negative correlation was found between serum vitamin D and CRP. Vitamin D 98-107 C-reactive protein Homo sapiens 112-115 30070873-10 2018 Individuals with inadequacy of vitamin D showed higher values of CRP in both groups (p = 0.000). Vitamin D 31-40 C-reactive protein Homo sapiens 65-68 30854952-0 2019 The Effects of Vitamin D Supplementation on Glycemic Control, Lipid Profiles and C-Reactive Protein Among Patients with Cardiovascular Disease: a Systematic Review and Meta-Analysis of Randomized Controlled Trials. Vitamin D 15-24 C-reactive protein Homo sapiens 81-99 30854952-3 2019 This systematic review and meta-analysis aimed to determine the effects of vitamin D supplementation on glycemic control, lipid profiles, and C-reactive protein among patients with coronary artery disease. Vitamin D 75-84 C-reactive protein Homo sapiens 142-160 30854952-10 2019 Additionally, vitamin D supplementation significantly reduced C-reactive protein (CRP) levels (WMD: -0.75; 95% CI: -1.28, -0.23). Vitamin D 14-23 C-reactive protein Homo sapiens 62-80 30854952-10 2019 Additionally, vitamin D supplementation significantly reduced C-reactive protein (CRP) levels (WMD: -0.75; 95% CI: -1.28, -0.23). Vitamin D 14-23 C-reactive protein Homo sapiens 82-85 30854952-11 2019 CONCLUSION: This meta-analysis demonstrated the beneficial effects of vitamin D supplementation on improving glycemic control, HDL-cholesterol and CRP levels among patients with CVD, though it did not affect triglycerides, total- and LDL-cholesterol levels. Vitamin D 70-79 C-reactive protein Homo sapiens 147-150 30221147-10 2018 Conclusion: Vitamin D deficiency was common in all IBD patients, but more pronounced in CD patients, in whom it also showed a significant inverse association with inflammatory markers such as CRP and FC. Vitamin D 12-21 C-reactive protein Homo sapiens 192-195 29248753-0 2018 The study of vitamin D administration effect on CRP and Interleukin-6 as prognostic biomarkers of ventilator associated pneumonia. Vitamin D 13-22 C-reactive protein Homo sapiens 48-51 29248753-6 2018 Although C-Reactive protein (CRP) levels showed an improving trend in the vitamin D group, no significant difference between groups (P=0.12) was found. Vitamin D 74-83 C-reactive protein Homo sapiens 9-27 29248753-6 2018 Although C-Reactive protein (CRP) levels showed an improving trend in the vitamin D group, no significant difference between groups (P=0.12) was found. Vitamin D 74-83 C-reactive protein Homo sapiens 29-32 29320465-0 2018 CRP Genotypes Predict Increased Risk to Co-Present with Low Vitamin D and Elevated CRP in a Group of Healthy Black South African Women. Vitamin D 60-69 C-reactive protein Homo sapiens 0-3 32153865-0 2018 Impact of vitamin D supplementation on C-reactive protein; a systematic review and meta-analysis of randomized controlled trials. Vitamin D 10-19 C-reactive protein Homo sapiens 39-57 32153865-1 2018 Background: To evaluate the effect of vitamin D supplementation on C-reactive protein (CRP) through a systematic review and meta-analysis of randomized control trials (RCTs). Vitamin D 38-47 C-reactive protein Homo sapiens 67-85 32153865-1 2018 Background: To evaluate the effect of vitamin D supplementation on C-reactive protein (CRP) through a systematic review and meta-analysis of randomized control trials (RCTs). Vitamin D 38-47 C-reactive protein Homo sapiens 87-90 32153865-2 2018 Methods: PubMed-Medline, SCOPUS, Google Scholar and Web of Science databases were searched (up until April 2016) to identify RCTs evaluating the impact of vitamin D supplementation on CRP. Vitamin D 155-164 C-reactive protein Homo sapiens 184-187 32153865-8 2018 Pooling the data together indicated a non-significant decrease in CRP level following administration of vitamin D (weighted mean difference [WMD] -0.26(mg/l), (95% CI -0.75 to 0.22, N = 26 arms, heterogeneity p = 0.042; I2 54.2%). Vitamin D 104-113 C-reactive protein Homo sapiens 66-69 28841229-13 2018 The mean difference in hs-CRP was significantly lower in the vitamin D group (P = .045). Vitamin D 61-70 C-reactive protein Homo sapiens 26-29 27975188-0 2017 Vitamin D deficiency is associated with acute ischemic stroke, C-reactive protein, and short-term outcome. Vitamin D 0-9 C-reactive protein Homo sapiens 63-81 29945132-3 2018 METHODS: We searched randomized controlled trials from PubMed and the Cochrane Library in October 2017 and conducted a meta-analysis to evaluate the effectiveness of vitamin D supplementation on high-sensitivity C-reactive protein (hs-CRP), tumor necrosis factor-alpha (TNF-alpha) and interleukin-6 (IL-6). Vitamin D 166-175 C-reactive protein Homo sapiens 212-230 29945132-6 2018 The results indicated that the vitamin D supplementation significant decreased the hs-CRP level by 0.45 mug/mL, whereas the vitamin D supplementation did not influence the TNF-alpha and IL-6. Vitamin D 31-40 C-reactive protein Homo sapiens 86-89 29945132-7 2018 Subgroup analysis showed that vitamin D significantly lowered hs-CRP by 0.34 mug/mL among trials with a daily vitamin D dose <=4,000 IU and by 0.31 mug/mL among trials with time of vitamin D supplementation > 12 weeks. Vitamin D 30-39 C-reactive protein Homo sapiens 65-68 29945132-8 2018 CONCLUSIONS: Vitamin D supplementation is beneficial for the reduction of hs-CRP inT2DM subjects but does not have a significant influence on TNF-alpha and IL-6 in T2DM subjects. Vitamin D 13-22 C-reactive protein Homo sapiens 77-80 29118583-1 2017 Objective: To determine if vitamin D deficiency was associated with higher odds of left ventricular dysfunction among patients with acute coronary syndrome (ACS) and, if so, to determine whether this association was mediated by increased inflammation as measured by C-reactive protein (CRP) and white blood cell count (WBC). Vitamin D 27-36 C-reactive protein Homo sapiens 286-289 28425575-0 2017 High Dose Supplementation of Vitamin D Affects Measures of Systemic Inflammation: Reductions in High Sensitivity C-Reactive Protein Level and Neutrophil to Lymphocyte Ratio (NLR) Distribution. Vitamin D 29-38 C-reactive protein Homo sapiens 113-131 28425575-3 2017 The aim of the current study was to evaluate the effect of vitamin D supplementation on serum C-Reactive Protein (CRP) level and Neutrophil-to-lymphocyte ratio (NLR) distribution in a large cohort of adolescent girls. Vitamin D 59-68 C-reactive protein Homo sapiens 94-112 28425575-3 2017 The aim of the current study was to evaluate the effect of vitamin D supplementation on serum C-Reactive Protein (CRP) level and Neutrophil-to-lymphocyte ratio (NLR) distribution in a large cohort of adolescent girls. Vitamin D 59-68 C-reactive protein Homo sapiens 114-117 28425575-8 2017 Interestingly we observed a significant reduction in neutrophil count and CRP level after high dose vitamin D supplementation. Vitamin D 100-109 C-reactive protein Homo sapiens 74-77 28425575-9 2017 Our findings showed that the high dose supplementation of vitamin D affects measures of systemic inflammation: reductions in High Sensitivity C-Reactive Protein level and Neutrophil-to-lymphocyte ratio (NLR) distribution. Vitamin D 58-67 C-reactive protein Homo sapiens 142-160 28382079-0 2017 The Relationship between Vitamin D and Coronary Artery Ectasia in Subjects with a Normal C-Reactive Protein Level. Vitamin D 25-34 C-reactive protein Homo sapiens 89-107 26778547-0 2016 The association between vitamin D and C-reactive protein levels in patients with inflammatory and non-inflammatory diseases. Vitamin D 24-33 C-reactive protein Homo sapiens 38-56 27555076-0 2016 Vitamin D Treatment Effect on Serum Endocan and High-Sensitivity C-Reactive Protein Levels in Renal Transplant Patients. Vitamin D 0-9 C-reactive protein Homo sapiens 65-83 27555076-3 2016 OBJECTIVE: Our aim was to investigate the relationship between vitamin D treatment and serum endocan and high-sensitivity C-reactive protein (hs-CRP) levels as inflammatory markers in transplant patients. Vitamin D 63-72 C-reactive protein Homo sapiens 122-140 27827910-1 2016 The hypothesized effect of vitamin D on C-reactive protein (CRP) has received substantial attention as a potential means to alleviate the risk for cardiovascular disease. Vitamin D 27-36 C-reactive protein Homo sapiens 40-58 27827910-1 2016 The hypothesized effect of vitamin D on C-reactive protein (CRP) has received substantial attention as a potential means to alleviate the risk for cardiovascular disease. Vitamin D 27-36 C-reactive protein Homo sapiens 60-63 27570748-1 2015 BACKGROUND: To investigate the effect of supplementation of standard treatment (inhaled long-acting beta2 agonists, anticholinergics and corticosteroids) with vitamin D on C reactive protein and pulmonary function tests in patients with COPD exacerbation. Vitamin D 159-168 C-reactive protein Homo sapiens 172-190 26873242-0 2016 The effect of a single dose of vitamin D on glycemic status and C-reactive protein levels in type 2 diabetic patients with ischemic heart disease: a randomized clinical trial. Vitamin D 31-40 C-reactive protein Homo sapiens 64-82 27798347-0 2016 Associations between Vitamin D and Cardiovascular Disease Risk Factors in African Americans Are Partly Explained by Circulating Adipokines and C-Reactive Protein: The Jackson Heart Study. Vitamin D 21-30 C-reactive protein Homo sapiens 143-161 27798347-11 2016 CONCLUSIONS: Our findings suggest that the associations between vitamin D status and CVD risk factors in AAs are partially mediated through circulating adipokines and CRP. Vitamin D 64-73 C-reactive protein Homo sapiens 167-170 27327576-7 2016 Binary logistic regression analysis showed a strong association between the presence of vitamin D deficiency and benign prostatic hyperplasia (BPH) after adjusting for age, International Prostate Symptom Score, urination time, urinary volume, abdominal obesity, aldosterone, glucose, insulin, parathyroid hormone, and C-reactive protein (odds ratio 5.22, 95% confidence interval 1.96-12.76, P = .001). Vitamin D 88-97 C-reactive protein Homo sapiens 318-336 27272805-6 2016 C-reactive protein (CRP) and interleukin-6 (IL-6) correlated with each other and exhibited positive correlation with age, body-mass index (BMI), leukocyte count, platelet count, kynurenine, kynurenine/tryptophan ratio and urinary neopterin and a negative correlation with vitamin D and retinol. Vitamin D 272-281 C-reactive protein Homo sapiens 0-18 27247730-0 2016 Effects of vitamin D supplementation on metabolic indices and hs-CRP levels in gestational diabetes mellitus patients: a randomized, double-blinded, placebo-controlled clinical trial. Vitamin D 11-20 C-reactive protein Homo sapiens 65-68 27247730-2 2016 This study evaluated the effect of vitamin D supplementation on metabolic indices and hs-C-reactive protein (CRP) levels in GDM patients. Vitamin D 35-44 C-reactive protein Homo sapiens 86-107 27001565-0 2016 Association of Vitamin D Levels With Outcome in Patients With Melanoma After Adjustment For C-Reactive Protein. Vitamin D 15-24 C-reactive protein Homo sapiens 92-110 27001565-8 2016 A lower vitamin D was associated with the blood draw during fall/winter months (P < .001), older age (P = .001), increased CRP (P < .001), increased tumor thickness (P < .001), ulcerated tumor (P = .0105), and advanced melanoma stage (P = .0024). Vitamin D 8-17 C-reactive protein Homo sapiens 126-129 27001565-10 2016 The effect of vitamin D on these outcome measures persisted after adjustment for CRP and other covariates. Vitamin D 14-23 C-reactive protein Homo sapiens 81-84 27001565-13 2016 Although lower vitamin D was strongly associated with higher CRP, the associations of lower vitamin D with poorer OS, MSS, and DFS were independent of this association. Vitamin D 15-24 C-reactive protein Homo sapiens 61-64 26778547-7 2016 RESULTS: The correlation between (log) ln-25(OH) vitamin D and ln-CRP was highly significant (p<0.001) with a regression coefficient of -0.879. Vitamin D 49-58 C-reactive protein Homo sapiens 66-69 26310644-14 2015 Steroid treatment, small intestine involved disease and high C reactive protein (more than 8 mg/L) are risk factors of vitamin D deficiency in CD children. Vitamin D 119-128 C-reactive protein Homo sapiens 61-79 26800186-12 2016 Compared to patients with sufficient vitamin D, patients with deficient vitamin D more frequently exhibited diabetes, elevated C-reactive protein levels, and hospital-acquired infections upon ICU admission, and they more frequently developed acute kidney injury. Vitamin D 72-81 C-reactive protein Homo sapiens 127-145 26106052-0 2015 High levels of vitamin D in relation to reduced risk of schizophrenia with elevated C-reactive protein. Vitamin D 15-24 C-reactive protein Homo sapiens 84-102 26106052-2 2015 This study aimed to examine the hypothesis that vitamin D is inversely associated with C-reactive protein (CRP) in patients with schizophrenia, and high levels of vitamin D may be linked to reduced risk of schizophrenia with elevated CRP. Vitamin D 48-57 C-reactive protein Homo sapiens 87-105 26106052-2 2015 This study aimed to examine the hypothesis that vitamin D is inversely associated with C-reactive protein (CRP) in patients with schizophrenia, and high levels of vitamin D may be linked to reduced risk of schizophrenia with elevated CRP. Vitamin D 48-57 C-reactive protein Homo sapiens 107-110 27175089-7 2016 However, subjects with deficient levels of vitamin D and high CRP produced significantly higher levels of the proinflammatory cytokines (TNF-alpha and IL-8) as compared to subjects with low CRP levels with nondeficient and deficient levels of vitamin D. Vitamin D 243-252 C-reactive protein Homo sapiens 62-65 27175089-8 2016 Further, the anti-inflammatory/proinflammatory ratios suggest a role of vitamin D in maintaining an anti-inflammatory environment at low levels of CRP, an association that is weaker at high CRP levels in subjects with subclinical inflammatory situations. Vitamin D 72-81 C-reactive protein Homo sapiens 147-150 27076887-7 2016 RESULTS: Consumption of calcium-Vitamin D co-supplements resulted in a significant reduction of serum high-sensitivity C-reactive protein levels compared with placebo (-1856.8 +- 2657.7 vs. 707.1 +- 3139.4 mug/mL, P = 0.006). Vitamin D 32-41 C-reactive protein Homo sapiens 119-137 27713770-7 2016 Generally, Vit D levels were associated indirectly with leukocytes/ neutrophils number or with ESR, CRP, and Fibrinogen levels. Vitamin D 11-16 C-reactive protein Homo sapiens 100-103 26569292-0 2015 Inverse Correlation between Vitamin D and C-Reactive Protein in Newborns. Vitamin D 28-37 C-reactive protein Homo sapiens 42-60 26106052-2 2015 This study aimed to examine the hypothesis that vitamin D is inversely associated with C-reactive protein (CRP) in patients with schizophrenia, and high levels of vitamin D may be linked to reduced risk of schizophrenia with elevated CRP. Vitamin D 163-172 C-reactive protein Homo sapiens 234-237 26106052-8 2015 The proportions of patients significantly increased with increasing quartiles of CRP, while significantly decreased with increasing quartiles of 25(OH)D. Among individuals with high CRP, participants with high 25(OH)D have significantly lower proportion (adjusted OR =0.217, 95% CI 0.063, 0.751) of schizophrenia compared to those with low 25(OH)D. The evidence suggested that high levels of vitamin D may be linked to reduced risk of schizophrenia with elevated CRP. Vitamin D 392-401 C-reactive protein Homo sapiens 182-185 26106052-8 2015 The proportions of patients significantly increased with increasing quartiles of CRP, while significantly decreased with increasing quartiles of 25(OH)D. Among individuals with high CRP, participants with high 25(OH)D have significantly lower proportion (adjusted OR =0.217, 95% CI 0.063, 0.751) of schizophrenia compared to those with low 25(OH)D. The evidence suggested that high levels of vitamin D may be linked to reduced risk of schizophrenia with elevated CRP. Vitamin D 392-401 C-reactive protein Homo sapiens 182-185 26147588-3 2015 The aim of our study was to investigate whether vitamin D has an inhibitory effect on systemic inflammation by assessing the association between serum levels of vitamin D and C-reactive protein. Vitamin D 48-57 C-reactive protein Homo sapiens 175-193 26147588-3 2015 The aim of our study was to investigate whether vitamin D has an inhibitory effect on systemic inflammation by assessing the association between serum levels of vitamin D and C-reactive protein. Vitamin D 161-170 C-reactive protein Homo sapiens 175-193 26147588-7 2015 In conclusion, higher levels of Vitamin D are associated with lower levels of C-reactive protein. Vitamin D 32-41 C-reactive protein Homo sapiens 78-96 24327720-7 2014 An interaction between baseline 25(OH)D and vitamin D supplementation was observed for outcome change in log CRP (month 3-month 0; P for interaction = 0.04). Vitamin D 44-53 C-reactive protein Homo sapiens 109-112 26487877-0 2015 Effect of Vitamin D supplementation on symptoms and C-reactive protein in migraine patients. Vitamin D 10-19 C-reactive protein Homo sapiens 52-70 26487877-4 2015 So, in this study, we investigated the effect of Vitamin D supplementation on symptoms and C-reactive protein (CRP) among patients with migraine. Vitamin D 49-58 C-reactive protein Homo sapiens 91-109 26487877-4 2015 So, in this study, we investigated the effect of Vitamin D supplementation on symptoms and C-reactive protein (CRP) among patients with migraine. Vitamin D 49-58 C-reactive protein Homo sapiens 111-114 24797104-9 2015 Female gender and high C-reactive protein (CRP) were associated with vitamin D deficiency in anemic group. Vitamin D 69-78 C-reactive protein Homo sapiens 23-41 24797104-9 2015 Female gender and high C-reactive protein (CRP) were associated with vitamin D deficiency in anemic group. Vitamin D 69-78 C-reactive protein Homo sapiens 43-46 24918698-1 2014 Vitamin D might elicit protective effects against cardiovascular disease by decreasing the level of circulating high-sensitivity C-reactive protein (hs-CRP), an inflammatory marker. Vitamin D 0-9 C-reactive protein Homo sapiens 152-155 24918698-2 2014 Thus, we conducted a meta-analysis of randomized controlled trials to evaluate the association of vitamin D supplementation with circulating hs-CRP level. Vitamin D 98-107 C-reactive protein Homo sapiens 144-147 24918698-5 2014 The results of the meta-analysis of 10 trials involving a total of 924 participants showed that vitamin D supplementation significantly decreased the circulating hs-CRP level by 1.08 mg/L (95% CI, -2.13, -0.03), with the evidence of heterogeneity. Vitamin D 96-105 C-reactive protein Homo sapiens 165-168 24918698-8 2014 In summary, vitamin D supplementation is beneficial for the reduction of circulating hs-CRP. Vitamin D 12-21 C-reactive protein Homo sapiens 88-91 24622804-10 2014 Among women with complete pill counts (97% adherence), the mean decrease in CRP was 1.18 mg/mL (46%) in the vitamin D arm compared with 0.46 mg/mL (25%) in the placebo arm (P = 0.03). Vitamin D 108-117 C-reactive protein Homo sapiens 76-79 25396060-0 2014 Vitamin D deficiency is common and associated with increased C-reactive protein in children and young adults with lupus: an Atherosclerosis Prevention in Pediatric Lupus Erythematosus substudy. Vitamin D 0-9 C-reactive protein Homo sapiens 61-79 25336462-9 2015 Adding biomarkers to the models to test for mediation, the vitamin D effect on respiratory health was not a consequence of any single marker but was partially attenuated as a combined result of leukocytes, AP, and CRP. Vitamin D 59-68 C-reactive protein Homo sapiens 214-217 25215557-9 2014 Individuals who received joint calcium-vitamin D supplements tended to have a decrease in serum high-sensitivity C-reactive protein levels compared with placebo after controlling for baseline levels (-1.14 +- 0.25 vs 0.02 +- 0.24 ng/mL, P = .09). Vitamin D 39-48 C-reactive protein Homo sapiens 113-131 25489444-0 2014 Effect of Vitamin D Supplementation on C-reactive Protein in Patients with Nonalcoholic Fatty Liver. Vitamin D 10-19 C-reactive protein Homo sapiens 39-57 25627231-6 2014 Four of 7 studies found a significant negative correlation between vitamin D levels and Bath Ankylosing Spondylitis Index (BASDAI), erythrocyte sedimentation rate (ESR) and C-reactive protein (CRP). Vitamin D 67-76 C-reactive protein Homo sapiens 173-191 25627231-6 2014 Four of 7 studies found a significant negative correlation between vitamin D levels and Bath Ankylosing Spondylitis Index (BASDAI), erythrocyte sedimentation rate (ESR) and C-reactive protein (CRP). Vitamin D 67-76 C-reactive protein Homo sapiens 193-196 24484740-3 2014 AIM: To assess relationship between vitamin D status and C-reactive protein (CRP), a nonspecific inflammatory marker of CSU activity. Vitamin D 36-45 C-reactive protein Homo sapiens 57-75 24484740-3 2014 AIM: To assess relationship between vitamin D status and C-reactive protein (CRP), a nonspecific inflammatory marker of CSU activity. Vitamin D 36-45 C-reactive protein Homo sapiens 77-80 23799341-11 2013 Vitamin D level was also independently associated with high-sensitivity C reactive protein (beta = -0.143, P = 0.047). Vitamin D 0-9 C-reactive protein Homo sapiens 72-90 23927858-9 2013 A significant positive association between vitamin D level and gait speed was found only in those with high CRP in stratified analyses. Vitamin D 43-52 C-reactive protein Homo sapiens 108-111 23927858-11 2013 CONCLUSION: These findings provide evidence of a potential joint effect of vitamin D and CRP on gait speed, suggesting that evaluation and correction of vitamin D levels may be especially important in individuals with high CRP levels. Vitamin D 153-162 C-reactive protein Homo sapiens 89-92 24132976-2 2013 OBJECTIVE: This study was designed to assess the effects of vitamin D supplementation on metabolic profiles, high-sensitivity C-reactive protein, and biomarkers of oxidative stress in pregnant women with GDM. Vitamin D 60-69 C-reactive protein Homo sapiens 126-144 23403039-6 2013 Patients who took oral vitamin D supplementation had lower Crohn"s disease activity index (p<0.05) and C-reactive protein (p=0.07) than non-users. Vitamin D 23-32 C-reactive protein Homo sapiens 106-124 22704299-0 2012 Findings of a relation between vitamin D and C-reactive protein: concerns about methods used and conclusions drawn. Vitamin D 31-40 C-reactive protein Homo sapiens 45-63 21934326-7 2012 RESULTS: Lower serum vitamin D levels were found to be associated with prehypertension independent of potential confounders including body mass index (BMI), serum cholesterol, C-reactive protein and estimated glomerular filtration rate. Vitamin D 21-30 C-reactive protein Homo sapiens 176-194 21550088-8 2011 We also demonstrate correlations of vitamin D status with insulin sensitivity, HDL-C, and C-reactive protein in PCOS patients, which support the increasing evidence that vitamin D deficiency is associated with multiple metabolic risk factors in PCOS women. Vitamin D 36-45 C-reactive protein Homo sapiens 90-108 21550088-8 2011 We also demonstrate correlations of vitamin D status with insulin sensitivity, HDL-C, and C-reactive protein in PCOS patients, which support the increasing evidence that vitamin D deficiency is associated with multiple metabolic risk factors in PCOS women. Vitamin D 170-179 C-reactive protein Homo sapiens 90-108 20300755-11 2011 Serum 25-OH vitamin D levels were significantly negatively correlated with DAS28, CRP, and HAQ (respectively, r = -0.431, P = 0.000, r = -0.276, P = 0.026, and r = -0.267, P = 0.031). Vitamin D 12-21 C-reactive protein Homo sapiens 82-85 21350098-7 2011 There was a significant negative correlation between vitamin D levels and high-sensitivity C-reactive protein, NFkappaB activity, and TLR4 expression (P < .05). Vitamin D 53-62 C-reactive protein Homo sapiens 91-109 19415724-5 2009 Subjects with vitamin D deficiency [25(OH)D level less than 50 nmol/L (20 ng/mL)] had higher mean values of serum C-reactive protein (CRP) (2.40 mg/L vs. 0.84 mg/L, P = 0.02) and creatinine (1.75 mg/dL vs. 1.24 mg/dL, P = 0.03) and lower serum albumin values (3.12 g/dL vs. 3.39 g/dL, P = 0.003) compared to subjects without vitamin D deficiency. Vitamin D 14-23 C-reactive protein Homo sapiens 114-132 19415724-5 2009 Subjects with vitamin D deficiency [25(OH)D level less than 50 nmol/L (20 ng/mL)] had higher mean values of serum C-reactive protein (CRP) (2.40 mg/L vs. 0.84 mg/L, P = 0.02) and creatinine (1.75 mg/dL vs. 1.24 mg/dL, P = 0.03) and lower serum albumin values (3.12 g/dL vs. 3.39 g/dL, P = 0.003) compared to subjects without vitamin D deficiency. Vitamin D 14-23 C-reactive protein Homo sapiens 134-137 19415724-8 2009 Association of vitamin D deficiency with higher serum CRP, serum creatinine and ISS stage at time of diagnosis suggests that vitamin D deficiency may portend poorer outcomes in subjects with MM. Vitamin D 15-24 C-reactive protein Homo sapiens 54-57 18491455-4 2008 We evaluated the association between four Vitamin D Receptor polymorphisms (i.e. those identified by the restriction enzymes BsmI, ApaI, TaqI and FokI) and serum level of C-reactive protein in 88 hemodialysis patients routinely treated with active Vitamin D (calcitriol). Vitamin D 42-51 C-reactive protein Homo sapiens 171-189 22497530-5 2012 In this study, we explore the possible association between CRP and key factors pertaining to the vitamin D axis--in particular, VDR gene polymorphisms--in cancer patients with cachexia. Vitamin D 97-106 C-reactive protein Homo sapiens 59-62 21724580-5 2011 After 6 months of treatment, in the vitamin D(3) supplementation group, CRP decreased 32% overall (P = 0.11), 37% in men (P = 0.05), and 41% among non-nonsteroidal anti-inflammatory drug (NSAID) users (P = 0.05) relative to placebo. Vitamin D 36-45 C-reactive protein Homo sapiens 72-75 20362753-0 2010 Does vitamin D modulate asymmetric dimethylarginine and C-reactive protein concentrations? Vitamin D 5-14 C-reactive protein Homo sapiens 56-74 19390750-1 2009 OBJECTIVE: To elucidate the association between vitamin D status, C-reactive protein (CRP) and fibrinogen. Vitamin D 48-57 C-reactive protein Homo sapiens 86-89 19370277-13 2009 Patients with vitamin D deficiency had significant differences in body mass index (p=0.003), metabolic syndrome (p=0.05), high sensitive C-reactive protein (p=0.009), microalbuminuria (p=0.04), and glumerular filtration rate (p=0.02), compared to patients with sufficient vitamin D. Vitamin D 14-23 C-reactive protein Homo sapiens 137-155