PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28318037-3	2017	Molecular bypass therapy with the TK2 products, deoxycytidine monophosphate (dCMP) and deoxythymidine monophosphate (dTMP), prolongs the life span of Tk2-deficient (Tk2-/- ) mice by 2- to 3-fold.	Deoxycytidine Monophosphate	48-75	thymidine kinase 2, mitochondrial	Mus musculus	34-37	
28318037-3	2017	Molecular bypass therapy with the TK2 products, deoxycytidine monophosphate (dCMP) and deoxythymidine monophosphate (dTMP), prolongs the life span of Tk2-deficient (Tk2-/- ) mice by 2- to 3-fold.	Deoxycytidine Monophosphate	48-75	thymidine kinase 2, mitochondrial	Mus musculus	150-153	
28318037-3	2017	Molecular bypass therapy with the TK2 products, deoxycytidine monophosphate (dCMP) and deoxythymidine monophosphate (dTMP), prolongs the life span of Tk2-deficient (Tk2-/- ) mice by 2- to 3-fold.	Deoxycytidine Monophosphate	77-81	thymidine kinase 2, mitochondrial	Mus musculus	34-37	
28318037-3	2017	Molecular bypass therapy with the TK2 products, deoxycytidine monophosphate (dCMP) and deoxythymidine monophosphate (dTMP), prolongs the life span of Tk2-deficient (Tk2-/- ) mice by 2- to 3-fold.	Deoxycytidine Monophosphate	77-81	thymidine kinase 2, mitochondrial	Mus musculus	150-153	
28318037-7	2017	In contrast, dCMP+dTMP+THU therapy decreased life span of Tk2-/- animals compared to dCMP+dTMP.	Deoxycytidine Monophosphate	13-17	thymidine kinase 2, mitochondrial	Mus musculus	58-61	
24968719-3	2014	Assessment of 13-day-old Tk2(-/-) mice treated with dCMP+dTMP 200 mg/kg/day each (Tk2(-/-200dCMP/) (dTMP)) demonstrated that in mutant animals, the compounds raise dTTP concentrations, increase levels of mtDNA, ameliorate defects of mitochondrial respiratory chain enzymes, and significantly prolong their lifespan (34 days with treatment versus 13 days untreated).	Deoxycytidine Monophosphate	52-56	thymidine kinase 2, mitochondrial	Mus musculus	25-28	
24968719-3	2014	Assessment of 13-day-old Tk2(-/-) mice treated with dCMP+dTMP 200 mg/kg/day each (Tk2(-/-200dCMP/) (dTMP)) demonstrated that in mutant animals, the compounds raise dTTP concentrations, increase levels of mtDNA, ameliorate defects of mitochondrial respiratory chain enzymes, and significantly prolong their lifespan (34 days with treatment versus 13 days untreated).	Deoxycytidine Monophosphate	52-56	thymidine kinase 2, mitochondrial	Mus musculus	82-85