PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
15499081-9	2005	Dimethyl amiloride (0.1 mM), an inhibitor of the basolateral sodium-hydrogen exchanger 1, also inhibited CO(2)-augumented DBS, although S-3226, a specific inhibitor of apical sodium-hydrogen exchanger 3, did not.	co(2)	105-110	solute carrier family 9 member A1	Rattus norvegicus	61-88	
16831598-10	2006	CONCLUSIONS: The duodenal hyperemic response to luminal CO(2) is dependent on cytosolic and membrane-bound CA isoforms, NHE-1, and TRPV1.	co(2)	56-61	solute carrier family 9 member A1	Rattus norvegicus	120-125	
16831598-12	2006	NHE-1 activation preceding TRPV1 stimulation suggests that luminal CO(2) is sensed as H(+) in the subepithelium.	co(2)	67-72	solute carrier family 9 member A1	Rattus norvegicus	0-5	
14724181-2	2004	In TRCs perfused with CO(2)/HCO(3)(-)-free solution (pH 7.4), removal of basolateral Na(+) decreased pH(i) reversibly and zoniporide, a specific NHE-1 blocker, inhibited the Na(+)-induced changes in pH(i).	co(2)	22-27	solute carrier family 9 member A1	Rattus norvegicus	145-150