PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 15499081-9 2005 Dimethyl amiloride (0.1 mM), an inhibitor of the basolateral sodium-hydrogen exchanger 1, also inhibited CO(2)-augumented DBS, although S-3226, a specific inhibitor of apical sodium-hydrogen exchanger 3, did not. co(2) 105-110 solute carrier family 9 member A1 Rattus norvegicus 61-88 16831598-10 2006 CONCLUSIONS: The duodenal hyperemic response to luminal CO(2) is dependent on cytosolic and membrane-bound CA isoforms, NHE-1, and TRPV1. co(2) 56-61 solute carrier family 9 member A1 Rattus norvegicus 120-125 16831598-12 2006 NHE-1 activation preceding TRPV1 stimulation suggests that luminal CO(2) is sensed as H(+) in the subepithelium. co(2) 67-72 solute carrier family 9 member A1 Rattus norvegicus 0-5 14724181-2 2004 In TRCs perfused with CO(2)/HCO(3)(-)-free solution (pH 7.4), removal of basolateral Na(+) decreased pH(i) reversibly and zoniporide, a specific NHE-1 blocker, inhibited the Na(+)-induced changes in pH(i). co(2) 22-27 solute carrier family 9 member A1 Rattus norvegicus 145-150