PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 24407487-4 2014 The (GT)n and (TA)n dinucleotide variations in heme oxygenase 1 (HMOX1) and bilirubin UDP-glucuronosyl transferase (UGT1A1) gene promoters were determined by fragment analysis. Dinucleoside Phosphates 20-32 heme oxygenase 1 Homo sapiens 47-63 31411335-1 2020 Objectives The number of dinucleotide repeats (GT) nmodulate expression of heme oxygenase 1 (HMOX1), a stress response gene. Dinucleoside Phosphates 25-37 heme oxygenase 1 Homo sapiens 75-91 31411335-1 2020 Objectives The number of dinucleotide repeats (GT) nmodulate expression of heme oxygenase 1 (HMOX1), a stress response gene. Dinucleoside Phosphates 25-37 heme oxygenase 1 Homo sapiens 93-98 26173399-4 2015 The inducible HO isoform (HO-1) has two functional genetic polymorphisms: (GT)n dinucleotide repeats and A(-413)T SNP (rs2071746), both of which can affect its promoter activity. Dinucleoside Phosphates 80-92 heme oxygenase 1 Homo sapiens 26-30 25956277-3 2015 A (GT) n dinucleotide repeat at HO-1 promoter is a polymorphic region and modulates gene transcription and associated with some of diseases. Dinucleoside Phosphates 9-21 heme oxygenase 1 Homo sapiens 32-36 31120979-2 2019 Recently, an increased number of dinucleotide repeats in the HMOX1 gene were reported to associate with development of AKI in cardiac surgery. Dinucleoside Phosphates 33-45 heme oxygenase 1 Homo sapiens 61-66 24407487-4 2014 The (GT)n and (TA)n dinucleotide variations in heme oxygenase 1 (HMOX1) and bilirubin UDP-glucuronosyl transferase (UGT1A1) gene promoters were determined by fragment analysis. Dinucleoside Phosphates 20-32 heme oxygenase 1 Homo sapiens 65-70 16455537-3 2005 A length polymorphic variant of the HO-1 gene promoter region, comprising (GT)n dinucleotide repeats, is associated with altered levels of gene transcription: long (= 36 GT) repeats are associated with decreased HO-1. Dinucleoside Phosphates 80-92 heme oxygenase 1 Homo sapiens 36-40 22583702-1 2012 Heme oxygenase-1 (HMOX-1) is activated by oxidative stress, and gene responsiveness is reportedly determined by the number of dinucleotide (GT(n)) repeats in its highly polymorphic promoter region. Dinucleoside Phosphates 126-138 heme oxygenase 1 Homo sapiens 0-16 22583702-1 2012 Heme oxygenase-1 (HMOX-1) is activated by oxidative stress, and gene responsiveness is reportedly determined by the number of dinucleotide (GT(n)) repeats in its highly polymorphic promoter region. Dinucleoside Phosphates 126-138 heme oxygenase 1 Homo sapiens 18-24 21911884-3 2011 The (GT)n and (TA)n dinucleotide variations in HMOX1 and UGT1A1 gene promoters, respectively, were determined by fragment analysis in all subjects. Dinucleoside Phosphates 20-32 heme oxygenase 1 Homo sapiens 47-52 20121710-5 2010 However, the patients with short (<24 GT) dinucleotide repeat in the HO-1 gene promoter on either allele had significantly higher prolonged unconjugated hyperbilirubinaemia than the healthy newborns. Dinucleoside Phosphates 45-57 heme oxygenase 1 Homo sapiens 72-76 17391133-2 2007 A functional dinucleotide repeat (GT)(n) polymorphism, within the HO-1 promoter, regulates gene expression; a short number of repeats (S-allele <25) increases transcription. Dinucleoside Phosphates 13-25 heme oxygenase 1 Homo sapiens 66-70 21725851-1 2012 Heme oxygenase-1 (HO-1) which is a rate-limiting enzyme in heme degradation processes shows a dinucleotide GT repeat in the promoter that alters the level of gene transcription. Dinucleoside Phosphates 94-106 heme oxygenase 1 Homo sapiens 0-16 21725851-1 2012 Heme oxygenase-1 (HO-1) which is a rate-limiting enzyme in heme degradation processes shows a dinucleotide GT repeat in the promoter that alters the level of gene transcription. Dinucleoside Phosphates 94-106 heme oxygenase 1 Homo sapiens 18-22 19299217-5 2009 According to our previous study, AVF failure was associated with a longer dinucleotide (GT)n repeat (n > or = 30) in the promoter of the heme oxygenase-1 (HO-1) gene. Dinucleoside Phosphates 74-86 heme oxygenase 1 Homo sapiens 140-156 19299217-5 2009 According to our previous study, AVF failure was associated with a longer dinucleotide (GT)n repeat (n > or = 30) in the promoter of the heme oxygenase-1 (HO-1) gene. Dinucleoside Phosphates 74-86 heme oxygenase 1 Homo sapiens 158-162 18045827-2 2008 A functional dinucleotide repeat (GT)(n) polymorphism, within the HO-1 promoter, regulates HO-1 gene expression; a short number of repeats (S-allele <25) increases transcription. Dinucleoside Phosphates 13-25 heme oxygenase 1 Homo sapiens 66-70 18045827-2 2008 A functional dinucleotide repeat (GT)(n) polymorphism, within the HO-1 promoter, regulates HO-1 gene expression; a short number of repeats (S-allele <25) increases transcription. Dinucleoside Phosphates 13-25 heme oxygenase 1 Homo sapiens 91-95 17520317-2 2007 A (GT)n dinucleotide repeat in HO-1 promoter is polymorphic and modulates the transcriptional activity of the gene. Dinucleoside Phosphates 8-20 heme oxygenase 1 Homo sapiens 31-35 16313248-1 2005 BACKGROUND: A functional GT dinucleotide length polymorphism in the haem oxygenase-1 (HO-1) gene promoter is thought to be involved in the pathogenesis of cardiovascular disease. Dinucleoside Phosphates 28-40 heme oxygenase 1 Homo sapiens 68-90 16210136-2 2005 Inducibility of this enzyme is modulated by a (GT)n dinucleotide length polymorphism in the HO-1 gene promoter. Dinucleoside Phosphates 52-64 heme oxygenase 1 Homo sapiens 92-96 16455537-3 2005 A length polymorphic variant of the HO-1 gene promoter region, comprising (GT)n dinucleotide repeats, is associated with altered levels of gene transcription: long (= 36 GT) repeats are associated with decreased HO-1. Dinucleoside Phosphates 80-92 heme oxygenase 1 Homo sapiens 212-216 15084931-2 2004 Inducibility of this enzyme is modulated by a (GT)n dinucleotide length polymorphism in the HO-1 gene promoter. Dinucleoside Phosphates 52-64 heme oxygenase 1 Homo sapiens 92-96 15028349-3 2004 A dinucleotide repeat in the HO-1 gene promoter shows a length polymorphism that modulates HO-1 gene transcription. Dinucleoside Phosphates 2-14 heme oxygenase 1 Homo sapiens 29-33 15028349-3 2004 A dinucleotide repeat in the HO-1 gene promoter shows a length polymorphism that modulates HO-1 gene transcription. Dinucleoside Phosphates 2-14 heme oxygenase 1 Homo sapiens 91-95 15140586-2 2004 A (GT)n dinucleotide repeat polymorphism in the HO-1 promoter has been shown to modulate HO-1 gene expression. Dinucleoside Phosphates 8-20 heme oxygenase 1 Homo sapiens 48-52 14683741-3 2004 A dinucleotide GT repeat in the promotor region of human HO-1 gene shows a length polymorphism that modulates the level of gene transcription. Dinucleoside Phosphates 2-14 heme oxygenase 1 Homo sapiens 57-61 14683741-11 2004 These findings suggest the genetic contribution to stent restenosis and support the notion that the long dinucleotide GT repeat in promotor region may interfere with HO-1 gene transcription, leading to decreased vascular protection upon injury. Dinucleoside Phosphates 105-117 heme oxygenase 1 Homo sapiens 166-170 15140586-2 2004 A (GT)n dinucleotide repeat polymorphism in the HO-1 promoter has been shown to modulate HO-1 gene expression. Dinucleoside Phosphates 8-20 heme oxygenase 1 Homo sapiens 89-93 11718398-2 2001 A dinucleotide repeat in the promoter region of the HO-1 gene shows a length polymorphism that modulates the level of gene transcription. Dinucleoside Phosphates 2-14 heme oxygenase 1 Homo sapiens 52-56 11718398-7 2001 Patients with short (<25 GT) dinucleotide repeats in the HO-1 gene promoter on either allele had restenosis significantly less often than patients with longer (> or = 25 GT) dinucleotide repeats (p = 0.01). Dinucleoside Phosphates 32-44 heme oxygenase 1 Homo sapiens 60-64