PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23065572-2	2013	A potentially functional dinucleotide polymorphism, G4C14-to-A4T14, has been identified in the 5" untranslated region of exon 2 of the p73 gene, which may theoretically form a stem-loop structure and affect gene expression.	Dinucleoside Phosphates	25-37	tumor protein p73	Homo sapiens	135-138	
25097786-0	2014	Role of p73 Dinucleotide Polymorphism in Prostate Cancer and p73 Protein Isoform Balance.	Dinucleoside Phosphates	12-24	tumor protein p73	Homo sapiens	8-11	
25097786-3	2014	Here we address the potential association of a dinucleotide polymorphism (DNP) in exon 2 of the p73 gene with PCa risk/progression and discern any disruption of p73 protein isoforms levels in cells harboring a p73 DNP allele.	Dinucleoside Phosphates	47-59	tumor protein p73	Homo sapiens	96-99	
24339541-0	2013	Polymorphism at P21 codon 31 and dinucleotide polymorphism of P73 gene and susceptibility to bladder cancer in individuals from North India.	Dinucleoside Phosphates	33-45	tumor protein p73	Homo sapiens	62-65	
23065572-8	2013	These results suggest that the p73 G4C14-to-A4T14 dinucleotide polymorphism may play a role in the development of chronic HBV-infected HCC in the Chinese population, especially among women.	Dinucleoside Phosphates	50-62	tumor protein p73	Homo sapiens	31-34	
15578704-0	2005	Dinucleotide polymorphism of p73 gene is associated with a reduced risk of lung cancer in a Chinese population.	Dinucleoside Phosphates	0-12	tumor protein p73	Homo sapiens	29-32	
21976716-2	2012	The p73 G4C14-to-A4T14 dinucleotide polymorphism, consisting of two single-nucleotide polymorphisms in the non-coding region of exon 2 that are in complete linkage disequilibrium, has been extensively studied in association with cancer risk.	Dinucleoside Phosphates	23-35	tumor protein p73	Homo sapiens	4-7	
15578704-3	2005	A potentially functional dinucleotide polymorphism, G4C14-to-A4T14, has been identified in the 5" untranslated region (UTR) of exon 2 of the p73 gene, which may theoretically form a stem-loop structure and affect gene expression.	Dinucleoside Phosphates	25-37	tumor protein p73	Homo sapiens	141-144	
15578704-7	2005	These results suggest that this p73 dinucleotide polymorphism may have a role in lung cancer susceptibility in our study population.	Dinucleoside Phosphates	36-48	tumor protein p73	Homo sapiens	32-35	
15921610-5	2005	CONCLUSION: These results suggested that this p73 dinucleotide polymorphism might have had a role to play in the susceptibility of lung cancer.	Dinucleoside Phosphates	50-62	tumor protein p73	Homo sapiens	46-49