PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
6412136-1	1983	The mei-9 and mus(2)201 mutants of Drosophila melanogaster were identified as mutagen-sensitive mutants on the basis of larval hypersensitivity to methyl methanesulfonate and characterized as excision repair-deficient on the basis of a greatly reduced capacity to excise thymine dimers from cellular DNA.	Methyl Methanesulfonate	147-170	meiotic 9	Drosophila melanogaster	4-9	
6412136-0	1983	Drosophila mutations at the mei-9 and mus(2)201 loci which block excision of thymine dimers also block induction of unscheduled DNA synthesis by methyl methanesulfonate, ethyl methanesulfonate, N-methyl-N-nitrosourea, UV light and X-rays.	Methyl Methanesulfonate	145-168	meiotic 9	Drosophila melanogaster	28-33	
3007278-5	1986	Similarly, after treatment with MMS, MNNG and ENNG, w+(TE) was somatically more mutable in the mei-9 strain and less mutable in the mei-41 and mus-104 strains than in the repair-proficient strain.	Methyl Methanesulfonate	32-35	meiotic 9	Drosophila melanogaster	95-100	
3920517-8	1985	The order of hypermutability of AAs with mei-9L1 relative to mei-9+ is MMS greater than MNU greater than DMN = EMS greater than iPMS = ENU = DEN = ENNG.	Methyl Methanesulfonate	71-74	meiotic 9	Drosophila melanogaster	41-46	
3920517-10	1985	These mei-9L1/mei-9+ indices are: MMS = 7.6, MNU = 5.4, DMN = 2.4, EMS = 2.4 and iPMS = ENU = DEN = ENNG = 1.	Methyl Methanesulfonate	34-37	meiotic 9	Drosophila melanogaster	6-11	
6796408-4	1980	Results indicate that the mei-9a female is strong potentiator of chromosome loss induced by MMS and procarbazine in line with the effect recently shown for dimethylnitrosamine (DMN)-induced chromosome lesions (Zimmering, et al 1980).	Methyl Methanesulfonate	92-95	meiotic 9	Drosophila melanogaster	26-31	
6792536-0	1981	Review of the current status of the mei-9a test for chromosome loss in Drosophila melanogaster: an assay with radically improved detection capacity for chromosome lesions induced by methyl methanesulfonate (MMS), dimethylnitrosamine (DMN), and especially diethylnitrosamine (DEN) and procarbazine.	Methyl Methanesulfonate	182-205	meiotic 9	Drosophila melanogaster	36-41	
6792536-0	1981	Review of the current status of the mei-9a test for chromosome loss in Drosophila melanogaster: an assay with radically improved detection capacity for chromosome lesions induced by methyl methanesulfonate (MMS), dimethylnitrosamine (DMN), and especially diethylnitrosamine (DEN) and procarbazine.	Methyl Methanesulfonate	207-210	meiotic 9	Drosophila melanogaster	36-41	
118375-5	1979	Analysis of double-mutants reveals that the mei-9+ product functions in a different repair pathway of methyl methanesulfonate-induced damage than do the normal products of the mus(1)103, mus(1)104 and mus(1)107 loci.	Methyl Methanesulfonate	102-125	meiotic 9	Drosophila melanogaster	44-49	
826452-8	1976	Larvae which are hemizygous for either of the two mutants that define the mei-9 locus are hypersensitive to killing by the mutagens methyl methanesulfonate, nitrogen mustard and 2-acetylaminofluorene.	Methyl Methanesulfonate	132-155	meiotic 9	Drosophila melanogaster	74-79