PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 31737205-5 2019 Our results showed that CoQ10 treatment could significantly decrease the levels of oxidative products (MDA) and increase the activities of antioxidant enzymes (SOD and GSH) against oxidative stress, as well as decrease the levels of pro-apoptotic proteins (Bax and Caspase-3) and increase the levels of anti-apoptotic proteins (Bcl-2) against apoptosis after SCI. coenzyme Q10 24-29 BCL2, apoptosis regulator Rattus norvegicus 328-333 34939895-12 2021 Co-administration of CoQ10 resulted in significant improvement in the histopathological picture, with a significant decrease in caspase-3 and iNOS immunoexpression and downregulation of the Bax/Bcl-2 gene expression ratio. coenzyme Q10 21-26 BCL2, apoptosis regulator Rattus norvegicus 194-199 31582012-0 2019 Coenzyme Q10 protects hepatocytes from ischemia reperfusion-induced apoptosis and oxidative stress via regulation of Bax/Bcl-2/PUMA and Nrf-2/FOXO-3/Sirt-1 signaling pathways. coenzyme Q10 0-12 BCL2, apoptosis regulator Rattus norvegicus 121-126 18817789-10 2008 Oral administration of Co Q10 in a low dose 200 mg/kg/day (group III) or a high dose 600 mg/kg/day (group IV), resulted in amelioration of the mitochondrial induced apoptosis by dose-dependent restoration of striatal complex I activity, ATP levels with temperate increase in expression of Bcl-2 as well as decrease in catalepsy score. coenzyme Q10 23-29 BCL2, apoptosis regulator Rattus norvegicus 289-294