PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32454590-7	2017	Results: Adenosine-5"-N-ethyluronamide (NECA), a stable adenosine analogue, concentration- and time-dependently stimulates IL-6 gene expression in skeletal muscle cells.	Adenosine-5'-(N-ethylcarboxamide)	40-44	interleukin 6	Homo sapiens	123-127	
15769552-1	2005	As we have shown earlier, the stable adenosine analogue NECA (N6-(R)-phenylisopropyladenosine) stimulates IL-6 expression in the human astrocytoma cell line U373 MG via the A(2b) receptor.	Adenosine-5'-(N-ethylcarboxamide)	56-60	interleukin 6	Homo sapiens	106-110	
15472138-3	2005	Under the normoxic condition, adenosine and its stable analog, 5"-(N-ethylcarboxamido)-adenosine, via activation of A2B adenosine receptors, increased the release of interleukin (IL)-6 by 14-fold and induced the differentiation of human lung fibroblasts to myofibroblasts.	Adenosine-5'-(N-ethylcarboxamide)	63-96	interleukin 6	Homo sapiens	166-184	
15472138-4	2005	This latter effect of 5"-(N-ethylcarboxamido)-adenosine was abolished by an IL-6-neutralizing antibody.	Adenosine-5'-(N-ethylcarboxamide)	22-55	interleukin 6	Homo sapiens	76-80	
8627294-4	1996	Here we show that the mixed A1 and A2 agonist 5"-(N-ethylcarboxamido) adenosine (NECA) induces an increase in IL-6 mRNA levels and protein synthesis in the human astrocytoma cell line U373 MG.	Adenosine-5'-(N-ethylcarboxamide)	46-79	interleukin 6	Homo sapiens	110-114	
15571210-6	2004	The adenosine receptor agonist 5"N-ethylcarboxamido-adenosine (NECA) used to modify cytokine release in cultures of whole blood taken from the patients, depressed the release of tumour necrosis factor-alpha (TNFalpha), but failed to depress the release of interleukin-1b (IL-1b) or interleukin-6 (IL-6), a difference from earlier studies of healthy control subjects and, thus, a difference which may contribute to the disease activity.	Adenosine-5'-(N-ethylcarboxamide)	31-61	interleukin 6	Homo sapiens	282-295	
15571210-6	2004	The adenosine receptor agonist 5"N-ethylcarboxamido-adenosine (NECA) used to modify cytokine release in cultures of whole blood taken from the patients, depressed the release of tumour necrosis factor-alpha (TNFalpha), but failed to depress the release of interleukin-1b (IL-1b) or interleukin-6 (IL-6), a difference from earlier studies of healthy control subjects and, thus, a difference which may contribute to the disease activity.	Adenosine-5'-(N-ethylcarboxamide)	31-61	interleukin 6	Homo sapiens	297-301	
15571210-6	2004	The adenosine receptor agonist 5"N-ethylcarboxamido-adenosine (NECA) used to modify cytokine release in cultures of whole blood taken from the patients, depressed the release of tumour necrosis factor-alpha (TNFalpha), but failed to depress the release of interleukin-1b (IL-1b) or interleukin-6 (IL-6), a difference from earlier studies of healthy control subjects and, thus, a difference which may contribute to the disease activity.	Adenosine-5'-(N-ethylcarboxamide)	63-67	interleukin 6	Homo sapiens	282-295	
15571210-6	2004	The adenosine receptor agonist 5"N-ethylcarboxamido-adenosine (NECA) used to modify cytokine release in cultures of whole blood taken from the patients, depressed the release of tumour necrosis factor-alpha (TNFalpha), but failed to depress the release of interleukin-1b (IL-1b) or interleukin-6 (IL-6), a difference from earlier studies of healthy control subjects and, thus, a difference which may contribute to the disease activity.	Adenosine-5'-(N-ethylcarboxamide)	63-67	interleukin 6	Homo sapiens	297-301	
12855406-6	2004	NECA increased the release of interleukin-6 and monocyte chemotactic protein-1 proteins with EC50 values of 1.26 +/- 0.25 microM and 0.40 +/- 0.08 microM, respectively, and the maximal folds of induction were 20.8 +/- 1.7- and 6.4 +/- 0.7-fold, respectively.	Adenosine-5'-(N-ethylcarboxamide)	0-4	interleukin 6	Homo sapiens	30-43	
8627294-4	1996	Here we show that the mixed A1 and A2 agonist 5"-(N-ethylcarboxamido) adenosine (NECA) induces an increase in IL-6 mRNA levels and protein synthesis in the human astrocytoma cell line U373 MG.	Adenosine-5'-(N-ethylcarboxamide)	81-85	interleukin 6	Homo sapiens	110-114	
34258288-3	2021	Results: Adenosine-5"-N-ethyluronamide (NECA), a stable adenosine analogue, significantly stimulate inflammatory mediator (IL-6) (p < 0.001) and nuclear receptors (NR4A) (p < 0.05) and significantly modulate metabolic (PFK, LCAD, PGC-1alpha, and CPT1B) gene expressions in skeletal muscle cells (p < 0.05, p < 0.05, p < 0.001, and p < 0.01, respectively).	Adenosine-5'-(N-ethylcarboxamide)	40-44	interleukin 6	Homo sapiens	123-127