PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23894078-7	2014	The increase in phosphorylation of AMP-activated protein kinase (AMPK) and ERK1/2 were observed in rBMSCs cultured in beta-CS/beta-TCP composite extracts.	beta-tricalcium phosphate	126-134	mitogen-activated protein kinase 3	Homo sapiens	75-81	
31742679-0	2020	Mg2+ in beta-TCP/Mg-Zn composite enhances the differentiation of human bone marrow stromal cells into osteoblasts through MAPK-regulated Runx2/Osx.	beta-tricalcium phosphate	8-16	mitogen-activated protein kinase 3	Homo sapiens	122-126	
31742679-8	2020	In conclusion, Mg2+ in beta-TCP/Mg-Zn extract promotes the osteogenic differentiation of hBMSCs via MAPK-regulated Runx2/Osx interaction.	beta-tricalcium phosphate	23-31	mitogen-activated protein kinase 3	Homo sapiens	100-104	
25783501-12	2015	(beta-TCP)-Mg-3AI-1Zn may upregulate cell proliferation via Shc and ERK1/2 MAPK signaling in SaOS-2 osteoblasts grown on Mg alloy surface.	beta-tricalcium phosphate	1-9	mitogen-activated protein kinase 3	Homo sapiens	68-74	
23894078-8	2014	The ALP expression, calcium deposition, and ERK1/2 phosphorylation of rBMSCs, which was promoted by ions released from beta-CS/beta-TCP composites, were blocked by an AMPK inhibitor, Compound C. These results indicate that bioactive ions extracted from beta-CS/beta-TCP composites could stimulate the osteogenic differentiation of rBMSCs via the AMPK-Erk1/2 pathway.	beta-tricalcium phosphate	127-135	mitogen-activated protein kinase 3	Homo sapiens	44-50	
23894078-8	2014	The ALP expression, calcium deposition, and ERK1/2 phosphorylation of rBMSCs, which was promoted by ions released from beta-CS/beta-TCP composites, were blocked by an AMPK inhibitor, Compound C. These results indicate that bioactive ions extracted from beta-CS/beta-TCP composites could stimulate the osteogenic differentiation of rBMSCs via the AMPK-Erk1/2 pathway.	beta-tricalcium phosphate	127-135	mitogen-activated protein kinase 3	Homo sapiens	351-357