PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
22078782-11	2012	Moreover, DHT enhanced FOXO1 expression in parallel with increased SOD2 protein but not with SOD1.	Dihydrotestosterone	10-13	superoxide dismutase 2	Homo sapiens	67-71	
32327976-5	2020	Our findings showed that DHT treatment suppresses the expression of estrogen receptor beta (ERbeta) and superoxide dismutase 2 (SOD2) through AR-mediated hypermethylation on the ERbeta promoter, and BBR treatment suppresses AR expression through hypermethylation on the AR promoter.	Dihydrotestosterone	25-28	superoxide dismutase 2	Homo sapiens	104-126	
32327976-5	2020	Our findings showed that DHT treatment suppresses the expression of estrogen receptor beta (ERbeta) and superoxide dismutase 2 (SOD2) through AR-mediated hypermethylation on the ERbeta promoter, and BBR treatment suppresses AR expression through hypermethylation on the AR promoter.	Dihydrotestosterone	25-28	superoxide dismutase 2	Homo sapiens	128-132	
25398805-5	2014	RESULTS: After treatment with H2O2, DHT decreased tyrosine phosphorylation/transcriptional activity of STAT3 and promoter activity of MnSOD while E2 increased them.	Dihydrotestosterone	36-39	superoxide dismutase 2	Homo sapiens	134-139	
22172488-5	2012	Treatment with Tempol (4-hydroxy-2,2,6,6-tetramethylpiperidine-N-oxyl), a SOD mimetic, not only lowered cellular superoxide levels but also concomitantly attenuated AR transcriptional activity and AR target gene expression in a dose- and time-dependent manner, in the presence and absence of dihydrotestosterone, the major endogenous AR agonist.	Dihydrotestosterone	292-311	superoxide dismutase 2	Homo sapiens	74-77