PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28638736-9	2017	We further demonstrate that the activation of TFEB by hydroxypropyl-beta-cyclodextrin in macrophages suppressed their M2 polarization and tumor-promoting capacity, and that macrophage-specific TFEB overexpression inhibited breast tumor growth in mice.	2-Hydroxypropyl-beta-cyclodextrin	54-85	transcription factor EB	Mus musculus	46-50	
32354235-12	2020	Furthermore, we demonstrated that 2-hydroxypropyl-beta-cyclodextrin (HPbetaCD), a clinical agent used to enhance the solubility of drugs, activates TFEB and inhibits AAA formation and progression in mice.	2-Hydroxypropyl-beta-cyclodextrin	34-67	transcription factor EB	Mus musculus	148-152	
32354235-12	2020	Furthermore, we demonstrated that 2-hydroxypropyl-beta-cyclodextrin (HPbetaCD), a clinical agent used to enhance the solubility of drugs, activates TFEB and inhibits AAA formation and progression in mice.	2-Hydroxypropyl-beta-cyclodextrin	69-77	transcription factor EB	Mus musculus	148-152	
32354235-13	2020	Finally, we found that HPbetaCD inhibits AAA in a VSMC TFEB-dependent manner in mouse models.	2-Hydroxypropyl-beta-cyclodextrin	23-31	transcription factor EB	Mus musculus	55-59	
32354235-15	2020	TFEB activation by HPbetaCD may be a promising therapeutic strategy for the prevention and treatment of AAA.	2-Hydroxypropyl-beta-cyclodextrin	19-27	transcription factor EB	Mus musculus	0-4