PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
1467929-4	1992	Unilateral electrolytic or excitatory amino acid induced lesions of the basal forebrain, including the nucleus of the diagonal band, result in reductions of AChE staining throughout ipsilateral granular retrosplenial cortex; particularly noteworthy are the reductions in layer I and the deeper cortical layers.	Excitatory Amino Acids	27-48	acetylcholinesterase	Rattus norvegicus	157-161	
2322817-4	1990	Unilateral electrolytic or excitatory amino acid induced lesions of the basal forebrain result in reductions of AChE staining in ipsilateral visual cortex, particularly in layers IV and V. Electrolytic or scalpel lesions, placed in white matter underlying dorsal and lateral neocortex to interrupt basal forebrain projections to visual cortex, also reduce AChE staining in visual cortex.	Excitatory Amino Acids	27-48	acetylcholinesterase	Rattus norvegicus	112-116	
2322817-4	1990	Unilateral electrolytic or excitatory amino acid induced lesions of the basal forebrain result in reductions of AChE staining in ipsilateral visual cortex, particularly in layers IV and V. Electrolytic or scalpel lesions, placed in white matter underlying dorsal and lateral neocortex to interrupt basal forebrain projections to visual cortex, also reduce AChE staining in visual cortex.	Excitatory Amino Acids	27-48	acetylcholinesterase	Rattus norvegicus	356-360	
18588864-1	2008	The toxicity of organophosphorous (OP) nerve agents is attributed to their irreversible inhibition of acetylcholinesterase (AChE), which leads to excessive accumulation of acetylcholine (ACh) and is followed by the release of excitatory amino acids (EAA).	Excitatory Amino Acids	226-248	acetylcholinesterase	Rattus norvegicus	102-122	
18588864-1	2008	The toxicity of organophosphorous (OP) nerve agents is attributed to their irreversible inhibition of acetylcholinesterase (AChE), which leads to excessive accumulation of acetylcholine (ACh) and is followed by the release of excitatory amino acids (EAA).	Excitatory Amino Acids	226-248	acetylcholinesterase	Rattus norvegicus	124-128	
18588864-1	2008	The toxicity of organophosphorous (OP) nerve agents is attributed to their irreversible inhibition of acetylcholinesterase (AChE), which leads to excessive accumulation of acetylcholine (ACh) and is followed by the release of excitatory amino acids (EAA).	Excitatory Amino Acids	250-253	acetylcholinesterase	Rattus norvegicus	102-122	
18588864-1	2008	The toxicity of organophosphorous (OP) nerve agents is attributed to their irreversible inhibition of acetylcholinesterase (AChE), which leads to excessive accumulation of acetylcholine (ACh) and is followed by the release of excitatory amino acids (EAA).	Excitatory Amino Acids	250-253	acetylcholinesterase	Rattus norvegicus	124-128	
2900050-1	1988	The effect of excitatory amino acid antagonists on antagonist on neuropeptide Y (NPY) and cholinergic neurons in the striatum of the rat was studied by means of NPY immunocytochemistry, DFP histochemistry for acetylcholinesterase (AChE), and biochemical determinations of choline acetyltransferase (ChAT).	Excitatory Amino Acids	14-35	acetylcholinesterase	Rattus norvegicus	231-235