PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
7590666-9	1995	It is tempting to speculate that enhanced NF-kappa B expression caused by free radical production/oxidative stress may modulate liver injury, perhaps through an effect on cytotoxic cytokine synthesis.	Free Radicals	74-86	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	42-52	
14519659-5	2003	Stretch-induced activation of NF-kappaB involves activation of stretch-activated (SA) channels and the production of free radicals.	Free Radicals	117-130	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	30-39	
24376801-4	2013	Adding ferrous citrate (FC) complexes to the primary cultured mouse cerebral endothelial cells (CEC) to mimic the SAH conditions, we also showed that FC up-regulates NOS2 through nuclear translocation of NFkappaB induced by free radicals generation.	Free Radicals	224-237	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	204-212	
23029446-0	2012	Ferrous citrate up-regulates the NOS2 through nuclear translocation of NFkappaB induced by free radicals generation in mouse cerebral endothelial cells.	Free Radicals	91-104	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	71-79	
11133232-2	2001	Various stimuli, including ischemia, hypoxia, free radicals, cytokines, and lipopolysaccharide (LPS), activate NF-kappa B by inducing phosphorylation of I kappa B. Phosphorylation of serine residues at positions 32 and 36 is critical for ubiquitination and degradation of I kappa B alpha with consequent migration of NF-kappa B to the nucleus.	Free Radicals	46-59	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	111-121	
11133232-2	2001	Various stimuli, including ischemia, hypoxia, free radicals, cytokines, and lipopolysaccharide (LPS), activate NF-kappa B by inducing phosphorylation of I kappa B. Phosphorylation of serine residues at positions 32 and 36 is critical for ubiquitination and degradation of I kappa B alpha with consequent migration of NF-kappa B to the nucleus.	Free Radicals	46-59	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	317-327	
11018074-7	2000	These data strongly support the hypothesis that free radicals from NADPH oxidase in hepatic Kupffer cells play a predominant role in the pathogenesis of early alcohol-induced hepatitis by activating NF-kappaB, which activates production of cytotoxic TNF-alpha.	Free Radicals	48-61	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	199-208