PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
12881227-7	2004	Nevertheless, coperfusion with 0.1 and 0.3 mM 5-nitro-2-(3-phenylpropylamino) benzoic acid, which inhibits the cystic fibrosis transmembrane conductor regulator (CFTR), dose dependently inhibited S3226-induced DBS.	dbs	210-213	CF transmembrane conductance regulator	Rattus norvegicus	111-160	
20805305-9	2010	Furthermore, ADO-induced DBS was enhanced by 2"-deoxycoformycin (1 muM) and formycin B (0.1 mM), but not by S-(4-nitrobenzyl)-6-thioinosine (0.1 mM), and it was abolished by CFTR(inh)-172 pretreatment (1 mg/kg i.p).	dbs	25-28	CF transmembrane conductance regulator	Rattus norvegicus	174-178	
12881227-7	2004	Nevertheless, coperfusion with 0.1 and 0.3 mM 5-nitro-2-(3-phenylpropylamino) benzoic acid, which inhibits the cystic fibrosis transmembrane conductor regulator (CFTR), dose dependently inhibited S3226-induced DBS.	dbs	210-213	CF transmembrane conductance regulator	Rattus norvegicus	162-166	
12881227-9	2004	Because NHE3 inhibition-increased DBS was inhibited by an anion channel inhibitor and because reciprocal CFTR regulation has been previously shown between NHE3 and apical membrane anion transporters, we speculate that NHE3 inhibition increased DBS by altering anion transporter function.	dbs	34-37	CF transmembrane conductance regulator	Rattus norvegicus	105-109	
12881227-9	2004	Because NHE3 inhibition-increased DBS was inhibited by an anion channel inhibitor and because reciprocal CFTR regulation has been previously shown between NHE3 and apical membrane anion transporters, we speculate that NHE3 inhibition increased DBS by altering anion transporter function.	dbs	244-247	CF transmembrane conductance regulator	Rattus norvegicus	105-109