PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
15066018-6	2004	Pretreatment with capsaicin or hexamethonium, combination of both pretreatments or vagotomy reduced HCl-induced c-Fos expression by 54%, 66%, 63% and 68%, respectively.	Hexamethonium	31-44	Fos proto-oncogene, AP-1 transcription factor subunit	Rattus norvegicus	112-117	
10409218-4	1999	Atropine and hexamethonium partially decreased c-fos expression (banding vs. banding + atropine/hexamethonium: 700 +/- 67% vs. 400 +/- 67%, P < 0.05).	Hexamethonium	13-26	Fos proto-oncogene, AP-1 transcription factor subunit	Rattus norvegicus	47-52	
11447037-4	2001	Hexamethonium (20 mg/kg) also prevented 3-h cold exposure-induced myenteric Fos expression by 76-80%, whereas atropine or bretylium had no effect.	Hexamethonium	0-13	Fos proto-oncogene, AP-1 transcription factor subunit	Rattus norvegicus	76-79	
11292608-4	2001	Hexamethonium (20 mg/kg sc) prevented Fos expression by 90%, whereas atropine (2 mg/kg sc) had no effect.	Hexamethonium	0-13	Fos proto-oncogene, AP-1 transcription factor subunit	Rattus norvegicus	38-41	
12588467-6	2003	Pre-treatment with capsaicin or hexamethonium or a combination of both pre-treatments reduced HCl-induced c-Fos expression by 54, 66 and 63%, respectively.	Hexamethonium	32-45	Fos proto-oncogene, AP-1 transcription factor subunit	Rattus norvegicus	106-111	
10409218-4	1999	Atropine and hexamethonium partially decreased c-fos expression (banding vs. banding + atropine/hexamethonium: 700 +/- 67% vs. 400 +/- 67%, P < 0.05).	Hexamethonium	96-109	Fos proto-oncogene, AP-1 transcription factor subunit	Rattus norvegicus	47-52	
9583573-10	1998	The ganglionic blocking agent, hexamethonium, which blocks autonomic but not afferent pathways to the LUT, decreased c-fos expression by 50%.	Hexamethonium	31-44	Fos proto-oncogene, AP-1 transcription factor subunit	Rattus norvegicus	117-122	
8568687-4	1995	Refeeding of fasted rats induced a transient increase in c-fos mRNA abundance in gastric corpus and antrum that was sixfold within 15 min and declined within 4 h. The response was not mediated by gastrinergic or muscarinic cholinergic mechanisms; it was reduced but not abolished by hexamethonium.	Hexamethonium	283-296	Fos proto-oncogene, AP-1 transcription factor subunit	Rattus norvegicus	57-62	
1422856-7	1992	c-fos expression was substantially attenuated in the superficial gray layer of superior colliculus, medial terminal nucleus of the accessory optic tract, and the interpeduncular nucleus by pretreatment with the centrally acting nicotine antagonist mecamylamine, 5 mg/kg IP, but not with the peripherally acting antagonist hexamethonium, 4 mg/kg IP.	Hexamethonium	322-335	Fos proto-oncogene, AP-1 transcription factor subunit	Rattus norvegicus	0-5