PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
29374219-4	2018	TSA or SAHA inhibited vimentin, HDACs 1, 7 and 8, upregulated E-cadherin mRNA and protein levels in the PDAC cells, and time-dependently downregulated Oct-4, Sox-2, and Nanog, as well as inhibited PDAC tumorsphere formation.	trichostatin A	0-3	Nanog homeobox	Homo sapiens	169-174	
23024790-5	2012	Moreover, TSA treatment led to further increase in the expression of Sox2 and Nanog in PCa cells with EMT phenotype, which was associated with cancer stem-like cell (CSLC) characteristics consistent with increased cell motility.	trichostatin A	10-13	Nanog homeobox	Homo sapiens	78-83	
18203677-8	2008	Furthermore, our analysis identified several stem cell genes and pluripotency-associated genes that are induced by TSA/AzaC in neurosphere cells, including Cd34, Cd133, Oct4, Nanog, Klf4, Bex1, and the Dppa family members Dppa2, 3, 4, and 5.	trichostatin A	115-118	Nanog homeobox	Homo sapiens	175-180	
25866813-5	2015	Moreover, the NT blastocysts generated using TSA-treated ABM-MSCs exhibited significantly higher TCN and increased pluripotency extent measured with relative abundance of Oct4 and Nanog mRNAs as compared to the TSA-untreated group.	trichostatin A	45-48	Nanog homeobox	Homo sapiens	180-185	
24144739-8	2013	TSA down-regulated the mRNA expression of Nanog and Sox2 in the primary mammospheres.	trichostatin A	0-3	Nanog homeobox	Homo sapiens	42-47	
24144739-9	2013	CONCLUSION: TSA can partially inhibit the self-renewal of breast cancer stem cells through a mechanism involving the down-regulation of Nanog and Sox2 expression, indicating the value of combined treatments with low-dose TSA and other anticancer drugs to achieve maximum inhibition of breast cancer stem cell self-renewal.	trichostatin A	12-15	Nanog homeobox	Homo sapiens	136-141	
24144739-9	2013	CONCLUSION: TSA can partially inhibit the self-renewal of breast cancer stem cells through a mechanism involving the down-regulation of Nanog and Sox2 expression, indicating the value of combined treatments with low-dose TSA and other anticancer drugs to achieve maximum inhibition of breast cancer stem cell self-renewal.	trichostatin A	221-224	Nanog homeobox	Homo sapiens	136-141