PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
29560375-8	2018	Improving spiking and dendritic hyperexcitability through a combination of chlorzoxazone and baclofen produced sustained improvements in motor dysfunction in SCA1 mice (P < 0.01).	Baclofen	93-101	ataxin 1	Mus musculus	158-162	
27440721-4	2017	We also show that enhancement of mGluR signalling by a clinically available drug, baclofen, leads to improvement of motor performance in SCA1 mice.	Baclofen	82-90	ataxin 1	Mus musculus	137-141	
27440721-11	2017	Enhancement of mGluR signalling by baclofen, a clinically available GABAB receptor agonist, led to an improvement of motor performance in SCA1 mice and the improvement lasted ~1 week after a single application of baclofen.	Baclofen	35-43	ataxin 1	Mus musculus	138-142	
27440721-11	2017	Enhancement of mGluR signalling by baclofen, a clinically available GABAB receptor agonist, led to an improvement of motor performance in SCA1 mice and the improvement lasted ~1 week after a single application of baclofen.	Baclofen	213-221	ataxin 1	Mus musculus	138-142	
27440721-12	2017	Moreover, the restoration of motor performance in baclofen-treated SCA1 mice matched the functional recovery of mGluR-mediated slow synaptic currents and mGluR-dependent short- and long-term synaptic plasticity.	Baclofen	50-58	ataxin 1	Mus musculus	67-71