PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
24563318-0	2014	Lithium chloride inhibits TGF-beta1-induced myofibroblast transdifferentiation via PI3K/Akt pathway in cultured fibroblasts from Tenon"s capsule of the human eye.	Lithium Chloride	0-16	transforming growth factor beta 1	Homo sapiens	26-35	
32012313-8	2020	All these compounds inhibited LiCl-induced WNT/beta-catenin reporter activation to a similar extent, whereas ICG-001, IC-2-OTBS, and IC-2-F almost completely suppressed TGF-beta1-induced SMAD reporter activation without apparent cytotoxicity.	Lithium Chloride	30-34	transforming growth factor beta 1	Homo sapiens	169-178	
28606794-7	2017	Finally, lithium chloride, a GSK3 inhibitor, also attenuated the TGF-beta1-induced increase in alpha-SMA, COL1, and FN expression.	Lithium Chloride	9-25	transforming growth factor beta 1	Homo sapiens	65-74	
24563318-5	2014	LiCl treatment reduced expression of TGF-beta1-induced alpha-SMA expression in HTFs.	Lithium Chloride	0-4	transforming growth factor beta 1	Homo sapiens	37-46	
24563318-6	2014	LiCl also decreased Akt phosphorylation induced by TGF-beta1.	Lithium Chloride	0-4	transforming growth factor beta 1	Homo sapiens	51-60	
24563318-8	2014	Thus, LiCl induces the suppression of transdifferentiation stimulated by TGF-beta1 by the regulation of PI3K/Akt signaling in HTFs.	Lithium Chloride	6-10	transforming growth factor beta 1	Homo sapiens	73-82	
21310903-8	2011	Furthermore, TGF-beta1-induced TGFBIp expression decreased by 35% and 67% after treatment with 5 mM and 10 mM LiCl, respectively.	Lithium Chloride	110-114	transforming growth factor beta 1	Homo sapiens	13-22	
20348013-10	2010	Inhibiting Snail degradation by pre-treatment with lithium chloride (LiCl) led to a further decrease in E-cadherin expression in cells concurrently exposed to TGFbeta(1)+AngII, confirming that Snail is a repressor of E-cadherin.	Lithium Chloride	51-67	transforming growth factor beta 1	Homo sapiens	159-175	
20348013-10	2010	Inhibiting Snail degradation by pre-treatment with lithium chloride (LiCl) led to a further decrease in E-cadherin expression in cells concurrently exposed to TGFbeta(1)+AngII, confirming that Snail is a repressor of E-cadherin.	Lithium Chloride	69-73	transforming growth factor beta 1	Homo sapiens	159-175	
17623674-8	2007	Lithium chloride, a GSK-3 inhibitor, increased expression of beta-catenin and attenuated TGF-beta1-dependent Smad3 signaling.	Lithium Chloride	0-16	transforming growth factor beta 1	Homo sapiens	89-98	
18346908-9	2008	Similarly to TGF-beta1, the GSK-3beta inhibitor lithium chloride (10mM), increased the total levels of beta-catenin and promoted alpha-SMA expression and collagen production.	Lithium Chloride	48-64	transforming growth factor beta 1	Homo sapiens	13-22	
16924593-9	2006	Significantly, the effects of LiCl also extended to blocking the potent EMT/cataract-promoting effects of transforming growth factor beta (TGFbeta) on lens epithelial cells.	Lithium Chloride	30-34	transforming growth factor beta 1	Homo sapiens	106-137	
16924593-9	2006	Significantly, the effects of LiCl also extended to blocking the potent EMT/cataract-promoting effects of transforming growth factor beta (TGFbeta) on lens epithelial cells.	Lithium Chloride	30-34	transforming growth factor beta 1	Homo sapiens	139-146