PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23979838-10	2013	Finally, we found that administration of lithium chloride, a well-known GSK-3beta inhibitor, resulted in decreased levels of phosphorylated tau in DJ-1L166P-transfected cells.	Lithium Chloride	41-57	microtubule associated protein tau	Homo sapiens	140-143	
32692785-7	2020	We found that TBB (a casein kinase II inhibitor), AR and LiCl (GSK-3 inhibitors), cyclosporin A (calcineurin inhibitor), and Saracatinib (Fyn kinase inhibitor) caused robust inhibition of OA-induced monomeric and oligomeric p-tau in both N2a and CTX culture.	Lithium Chloride	57-61	microtubule associated protein tau	Homo sapiens	226-229	
30448465-0	2019	Pore-former enabled seeding of tau in rats: Alleviation by memantine and lithium chloride.	Lithium Chloride	73-89	microtubule associated protein tau	Homo sapiens	31-34	
20032300-4	2010	Furthermore, adding synthetic Abeta peptide, the pathogenic product of APP, to cultured cells causes rapid and robust chromosome mis-segregation leading to aneuploid, including trisomy 21, daughters, which is prevented by LiCl addition or Ca(2+) chelation and is replicated in tau KO cells, implicating GSK-3beta, calpain, and Tau-dependent microtubule transport in the aneugenic activity of Abeta.	Lithium Chloride	222-226	microtubule associated protein tau	Homo sapiens	277-280	
21561378-6	2011	The concentration-dependent reduction of tau phosphorylation by GSK-3 inhibitors, LiCl, Chir98023, and SB415286, was examined by immunoblot analysis and Tau ELISA (in situ ELISA).	Lithium Chloride	82-86	microtubule associated protein tau	Homo sapiens	41-44	
20617325-6	2010	These deleterious effects of human tau are phosphorylation dependent because treatment with LiCl to suppress tau phosphorylation increases microtubule binding of both human and Drosophila tau and restores cytoskeletal integrity.	Lithium Chloride	92-96	microtubule associated protein tau	Homo sapiens	35-38	
20617325-6	2010	These deleterious effects of human tau are phosphorylation dependent because treatment with LiCl to suppress tau phosphorylation increases microtubule binding of both human and Drosophila tau and restores cytoskeletal integrity.	Lithium Chloride	92-96	microtubule associated protein tau	Homo sapiens	109-112	
20617325-6	2010	These deleterious effects of human tau are phosphorylation dependent because treatment with LiCl to suppress tau phosphorylation increases microtubule binding of both human and Drosophila tau and restores cytoskeletal integrity.	Lithium Chloride	92-96	microtubule associated protein tau	Homo sapiens	109-112	
19782075-9	2010	Treatment of tau(wt)/Abeta(42) and flies with LiCl ameliorates the exacerbating effect of Abeta(42), suggesting that GSK-3beta may be involved in the mechanism by which Abeta(42) and tau(wt) interact to cause neuronal dysfunction.	Lithium Chloride	46-50	microtubule associated protein tau	Homo sapiens	183-186	
20032300-4	2010	Furthermore, adding synthetic Abeta peptide, the pathogenic product of APP, to cultured cells causes rapid and robust chromosome mis-segregation leading to aneuploid, including trisomy 21, daughters, which is prevented by LiCl addition or Ca(2+) chelation and is replicated in tau KO cells, implicating GSK-3beta, calpain, and Tau-dependent microtubule transport in the aneugenic activity of Abeta.	Lithium Chloride	222-226	microtubule associated protein tau	Homo sapiens	327-330	
19429119-7	2009	This was based on the following: Leptin and the cell-permeable AMPK activator, 5-aminoimidazole-4-carboxyamide ribonucleoside (AICAR), reduced tau phosphorylation at AD-relevant sites similarly to the GSK-3beta inhibitor, lithium chloride (LiCl).	Lithium Chloride	222-238	microtubule associated protein tau	Homo sapiens	143-146	
19429119-7	2009	This was based on the following: Leptin and the cell-permeable AMPK activator, 5-aminoimidazole-4-carboxyamide ribonucleoside (AICAR), reduced tau phosphorylation at AD-relevant sites similarly to the GSK-3beta inhibitor, lithium chloride (LiCl).	Lithium Chloride	240-244	microtubule associated protein tau	Homo sapiens	143-146	
16257959-4	2005	Our first finding that treatment of cells with LiCl, a selective inhibitor of another major tau kinase, glycogen synthase kinase-3beta (GSK-3beta), inhibits phosphorylation of Ser-262 of tau led us to investigate the possible involvement of GSK-3beta in MARK2 activation.	Lithium Chloride	47-51	microtubule associated protein tau	Homo sapiens	92-95	
16257959-4	2005	Our first finding that treatment of cells with LiCl, a selective inhibitor of another major tau kinase, glycogen synthase kinase-3beta (GSK-3beta), inhibits phosphorylation of Ser-262 of tau led us to investigate the possible involvement of GSK-3beta in MARK2 activation.	Lithium Chloride	47-51	microtubule associated protein tau	Homo sapiens	187-190	
11602330-7	2001	The bundles induced by GSK-3beta phosphorylated tau were sensitive to colchicine, and could be reversed by the addition of LiCl, an inhibitor of GSK-3beta.	Lithium Chloride	123-127	microtubule associated protein tau	Homo sapiens	48-51