PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 28721338-7 2017 The beneficial action of EPA and DHA reported in the animal experimental models of AAA induced by Ang-II infusion can be attributed to their (EPA and DHA) ability to enhance the formation of not only resolvins, protectins and maresins but also LXA4. Eicosapentaenoic Acid 25-28 angiotensinogen Homo sapiens 98-104 28721338-7 2017 The beneficial action of EPA and DHA reported in the animal experimental models of AAA induced by Ang-II infusion can be attributed to their (EPA and DHA) ability to enhance the formation of not only resolvins, protectins and maresins but also LXA4. Eicosapentaenoic Acid 142-145 angiotensinogen Homo sapiens 98-104 12944026-8 2003 Eicosapentaenoic acid (10 or 25 micromol/L) did not alter basal intracellular calcium concentration, but decreased the peak response to 100 nmol/L angiotensin II. Eicosapentaenoic Acid 0-21 angiotensinogen Homo sapiens 147-161 22475809-3 2012 Here, we determined differential effects of eicosapentaenoic acid (EPA, n-3 PUFA) vs. arachidonic acid (AA, n-6 PUFA) on expression and secretion of angiotensinogen (Agt), interleukin 6 (IL-6) and monocyte chemotactic protein (MCP-1) in 3T3-L1 adipocytes. Eicosapentaenoic Acid 44-65 angiotensinogen Homo sapiens 149-164 22475809-3 2012 Here, we determined differential effects of eicosapentaenoic acid (EPA, n-3 PUFA) vs. arachidonic acid (AA, n-6 PUFA) on expression and secretion of angiotensinogen (Agt), interleukin 6 (IL-6) and monocyte chemotactic protein (MCP-1) in 3T3-L1 adipocytes. Eicosapentaenoic Acid 67-70 angiotensinogen Homo sapiens 149-164 22475809-4 2012 While both PUFAs increased intracellular Agt protein and mRNA expression, Agt secretion into culture media was increased only by AA treatment, which in turn was prevented by co-treatment with EPA. Eicosapentaenoic Acid 192-195 angiotensinogen Homo sapiens 74-77 22475809-6 2012 Moreover, IL-6 and MCP-1 were more significantly reduced by EPA treatment compared to Agt (IL-6>MCP>Agt). Eicosapentaenoic Acid 60-63 angiotensinogen Homo sapiens 106-109 19229829-0 2009 EPA and DHA suppress AngII- and arachidonic acid-induced expression of profibrotic genes in human mesangial cells. Eicosapentaenoic Acid 0-3 angiotensinogen Homo sapiens 21-26 15610248-3 2005 As nitric oxide production via iNOS can inhibit CYP AA metabolism, we tested the hypothesis that by blocking iNOS or by supplementing eicosapentanoic acid (EPA), which can serve as an alternative CYP substrate, Ang II-induced vasculopathy could be ameliorated. Eicosapentaenoic Acid 134-154 angiotensinogen Homo sapiens 211-217 15610248-3 2005 As nitric oxide production via iNOS can inhibit CYP AA metabolism, we tested the hypothesis that by blocking iNOS or by supplementing eicosapentanoic acid (EPA), which can serve as an alternative CYP substrate, Ang II-induced vasculopathy could be ameliorated. Eicosapentaenoic Acid 156-159 angiotensinogen Homo sapiens 211-217 12944026-9 2003 Eicosapentaenoic acid also decreased the amount of calcium released by thapsigargin, a drug that releases calcium from the sarcoplasmic reticulum, and decreased cation influx after angiotensin II stimulation. Eicosapentaenoic Acid 0-21 angiotensinogen Homo sapiens 181-195