PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
7535028-11	1995	Exposure of human umbilical vein endothelial cells (HUVEC) to EPA reduced the expression of VCAM-1, ELAM-1, and ICAM-1 after 5 hours of stimulation with LPS.	Eicosapentaenoic Acid	62-65	vascular cell adhesion molecule 1	Homo sapiens	92-98	
20112284-7	2010	In addition, pretreatment with ecosapentaenoic acid (EPA), a substrate competitor of arachidonic acid (AA), also blocked LPS-induced VCAM-1 mRNA and protein expression, and THP-1 adherence.	Eicosapentaenoic Acid	53-56	vascular cell adhesion molecule 1	Homo sapiens	133-139	
15514264-6	2004	Changes in CRP and VCAM-1 were inversely associated with changes in serum eicosapentaenoic acid (EPA) (r = -0.496, P = 0.016; r = -0.418, P = 0.047), or EPA plus docosapentaenoic acid (r = -0.409, P = 0.053; r = -0.357, P = 0.091) after subjects consumed the ALA Diet.	Eicosapentaenoic Acid	74-95	vascular cell adhesion molecule 1	Homo sapiens	19-25	
15514264-6	2004	Changes in CRP and VCAM-1 were inversely associated with changes in serum eicosapentaenoic acid (EPA) (r = -0.496, P = 0.016; r = -0.418, P = 0.047), or EPA plus docosapentaenoic acid (r = -0.409, P = 0.053; r = -0.357, P = 0.091) after subjects consumed the ALA Diet.	Eicosapentaenoic Acid	97-100	vascular cell adhesion molecule 1	Homo sapiens	19-25	
15514264-6	2004	Changes in CRP and VCAM-1 were inversely associated with changes in serum eicosapentaenoic acid (EPA) (r = -0.496, P = 0.016; r = -0.418, P = 0.047), or EPA plus docosapentaenoic acid (r = -0.409, P = 0.053; r = -0.357, P = 0.091) after subjects consumed the ALA Diet.	Eicosapentaenoic Acid	153-156	vascular cell adhesion molecule 1	Homo sapiens	19-25	
12204798-6	2002	A significant inverse linear association was found between serum non-esterified EPA and DHA, and soluble vascular cell adhesion molecule-1 (sVCAM-1) (P=0.02 and 0.001, respectively).	Eicosapentaenoic Acid	80-83	vascular cell adhesion molecule 1	Homo sapiens	105-138	
20573493-5	2011	DHA and eicosapentaenoic acid decreased VCAM-1 expression in a dose-dependent manner in TNF-alpha treated HAECs, while cis-linoleic acid and arachidonic acid did not have any significant effect on either VCAM-1 or ICAM-1 expression.	Eicosapentaenoic Acid	8-29	vascular cell adhesion molecule 1	Homo sapiens	40-46	
35065163-9	2022	Incubation of endothelial cells with ARA or EPA and BaP resulted in lower CYP1A1 and cPLA2 activities and lower expression of VCAM-1 and ICAM-1 genes.	Eicosapentaenoic Acid	44-47	vascular cell adhesion molecule 1	Homo sapiens	126-132	
30102930-6	2018	These studies indicate that n-3 PUFAs (both eicosapentaenoic acid and docosahexaenoic acid) can decrease the expression of key adhesion molecules, such as vascular cell adhesion molecule 1, by ECs and that this results in decreased adhesive interactions between leukocytes and ECs.	Eicosapentaenoic Acid	44-65	vascular cell adhesion molecule 1	Homo sapiens	155-188	
25631736-4	2015	Our results showed that n-3 PUFAs docosahexaenoic acid (DHA) and eicosapentaenoic acid (EPA) remarkably attenuated the adhesion of THP-1 cells to HAECs, probably through inhibiting the expression of VCAM-1 and ICAM-1.	Eicosapentaenoic Acid	65-86	vascular cell adhesion molecule 1	Homo sapiens	199-205	
25631736-4	2015	Our results showed that n-3 PUFAs docosahexaenoic acid (DHA) and eicosapentaenoic acid (EPA) remarkably attenuated the adhesion of THP-1 cells to HAECs, probably through inhibiting the expression of VCAM-1 and ICAM-1.	Eicosapentaenoic Acid	88-91	vascular cell adhesion molecule 1	Homo sapiens	199-205	
25463091-8	2014	In addition, the EPA-rich HDL significantly improved endothelial cell migration, and markedly inhibited cytokine-induced expression of vascular cell adhesion molecule-1, in human umbilical vein endothelial cells, compared to HDL before the EPA treatment.	Eicosapentaenoic Acid	17-20	vascular cell adhesion molecule 1	Homo sapiens	135-168