PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 21093158-0 2011 Differential effect of amphetamine on c-fos expression in female aromatase knockout (ArKO) mice compared to wildtype controls. Amphetamine 23-34 FBJ osteosarcoma oncogene Mus musculus 38-43 21093158-3 2011 This study investigates the effect of gonadal status on amphetamine-induced c-fos expression in the brains of female aromatase knockout and wildtype mice. Amphetamine 56-67 FBJ osteosarcoma oncogene Mus musculus 76-81 21093158-6 2011 Aromatase knockout mice showed markedly reduced amphetamine-induced Fos immunoreactivity compared to wildtype mice. Amphetamine 48-59 FBJ osteosarcoma oncogene Mus musculus 68-71 21093158-9 2011 These results indicate that mechanisms involved in amphetamine-induced c-fos expression are altered in aromatase knockout mice and that the primary hormone involved in this effect is not estrogen, but may be another factor released from the ovaries, such as an androgen. Amphetamine 51-62 FBJ osteosarcoma oncogene Mus musculus 71-76 21093158-10 2011 These results provide new insight into the effect of gonadal hormones on amphetamine induced c-fos expression in this mouse model of estrogen deficiency. Amphetamine 73-84 FBJ osteosarcoma oncogene Mus musculus 93-98 17276509-9 2007 Both HRs and LRs expressed amphetamine-induced sensitized locomotor activation and increased expression of Fos protein. Amphetamine 27-38 FBJ osteosarcoma oncogene Mus musculus 107-110 18184321-6 2008 Both stress- and amphetamine-pre-treated groups showed changes in amphetamine-induced Fos expression; however, none of these changes was shared by the two sensitizing treatments. Amphetamine 17-28 FBJ osteosarcoma oncogene Mus musculus 86-89 18184321-6 2008 Both stress- and amphetamine-pre-treated groups showed changes in amphetamine-induced Fos expression; however, none of these changes was shared by the two sensitizing treatments. Amphetamine 66-77 FBJ osteosarcoma oncogene Mus musculus 86-89 18185498-6 2008 Consistent with the behavioral findings, cocaine induces less c-Fos expression in the striatum of these mice, while amphetamine-induced c-Fos expression is intact. Amphetamine 116-127 FBJ osteosarcoma oncogene Mus musculus 136-141 15467708-0 2004 Amphetamine-induced Fos is reduced in limbic cortical regions but not in the caudate or accumbens in a genetic model of NMDA receptor hypofunction. Amphetamine 0-11 FBJ osteosarcoma oncogene Mus musculus 20-23 16632196-0 2006 Amphetamine-elicited striatal Fos expression is attenuated in neurotensin null mutant mice. Amphetamine 0-11 FBJ osteosarcoma oncogene Mus musculus 30-33 16687500-5 2006 c-Fos-positive neurons were increased in the prefrontal cortex in amphetamine-treated Adcyap1(-/-) mice, suggesting increased inhibitory control by prefrontal neurons. Amphetamine 66-77 FBJ osteosarcoma oncogene Mus musculus 0-5 15467708-9 2004 However, amphetamine-induced Fos was substantially attenuated in the medial frontal (infralimbic) and cingulate cortices, basolateral amygdala, and in the lateral septum of the mutant mice. Amphetamine 9-20 FBJ osteosarcoma oncogene Mus musculus 29-32 11978395-10 2002 The results of this study indicate that MPH induces the expression of c-fos in the same brain regions as cocaine and amphetamine, and that this expression is distributed differentially according to the age of the mouse. Amphetamine 117-128 FBJ osteosarcoma oncogene Mus musculus 70-75 15464745-2 2004 We used Fos expression as a tool to reveal strain differences in the postsynaptic effects of amphetamine (AMPH; 2.5 mg/kg) within the nucleus accumbens (NAc) (core and shell) and the dorsal caudate (dorsomedial and dorsolateral). Amphetamine 93-104 FBJ osteosarcoma oncogene Mus musculus 8-11 15464745-3 2004 AMPH stimulated Fos expression in all striatal regions of mice from both strains. Amphetamine 0-4 FBJ osteosarcoma oncogene Mus musculus 16-19 16713106-0 2006 Habituation to the test cage influences amphetamine-induced locomotion and Fos expression and increases FosB/DeltaFosB-like immunoreactivity in mice. Amphetamine 40-51 FBJ osteosarcoma oncogene Mus musculus 75-78 16713106-5 2006 Moreover, previous habituation to the test cage reduced the locomotor response to a low dose of amphetamine only in DBA mice while it reduced amphetamine-induced Fos expression in medial-prefrontal cortex, dorsal caudate and the accumbens shell of mice of the C57 strain. Amphetamine 142-153 FBJ osteosarcoma oncogene Mus musculus 162-165 16713106-7 2006 Moreover, they suggest that the procedure of daily familiarization influences the pattern of brain Fos expression induced by amphetamine. Amphetamine 125-136 FBJ osteosarcoma oncogene Mus musculus 99-102 35165173-8 2022 The deletion led to alterations in several behavioral domains, namely to an increased anxiety-like behavior, decrease in sociability ratio, deficit in discrimination learning and increased amphetamine-induced hyperlocomotion and c-Fos expression in mice with beta2 deletion. Amphetamine 189-200 FBJ osteosarcoma oncogene Mus musculus 229-234 31721208-5 2020 Moreover, amphetamine-induced elevations of c-Fos expression and dopamine level in the striatum were greater in Het mice than in WT controls, suggesting an altered dopaminergic regulation in the striatum of Het mice. Amphetamine 10-21 FBJ osteosarcoma oncogene Mus musculus 44-49 28857482-6 2018 In addition, Cdkn1cBACx1 animals were hypersensitive to amphetamine as showed by c-fos expression in the nucleus accumbens. Amphetamine 56-67 FBJ osteosarcoma oncogene Mus musculus 81-86 29394409-6 2019 Interestingly, the magnitude and distribution of amphetamine-induced c-Fos expression in dopamine neurons was comparable between genotypes across dopaminergic input subregions in the ventral tegmental area (VTA). Amphetamine 49-60 FBJ osteosarcoma oncogene Mus musculus 69-74 28840858-3 2018 AMPH induced significantly more expression of the activity-dependent gene Fos in both D1 and D2 dopamine receptor-expressing medium spiny neurons (MSNs) of the NAc of PV+ interneuron silenced mice, suggesting a function for PV+ interneuron-mediated MSN inhibition in the expression of AMPH-induced locomotor sensitization and CPP. Amphetamine 0-4 FBJ osteosarcoma oncogene Mus musculus 74-77 28840858-3 2018 AMPH induced significantly more expression of the activity-dependent gene Fos in both D1 and D2 dopamine receptor-expressing medium spiny neurons (MSNs) of the NAc of PV+ interneuron silenced mice, suggesting a function for PV+ interneuron-mediated MSN inhibition in the expression of AMPH-induced locomotor sensitization and CPP. Amphetamine 285-289 FBJ osteosarcoma oncogene Mus musculus 74-77 24574986-10 2014 c-Fos quantification in the nucleus accumbens further confirmed defective neuronal activation following amphetamine injection. Amphetamine 104-115 FBJ osteosarcoma oncogene Mus musculus 0-5