PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31523846-4	2019	The aim of this study was to investigate A1AT serum levels and the rs1303 (Pi*M3) variant in A1AT gene in patients with ASA.	Aspirin	120-123	serpin family A member 1	Homo sapiens	41-45	
31523846-4	2019	The aim of this study was to investigate A1AT serum levels and the rs1303 (Pi*M3) variant in A1AT gene in patients with ASA.	Aspirin	120-123	serpin family A member 1	Homo sapiens	93-97	
31523846-10	2019	A1AT homozygote mutation (PiM3M3) was significantly higher in the ASA group than the control group (21 vs 11, OR (95% CI): 6.68 [2.09-21.40], P = .001).	Aspirin	66-69	serpin family A member 1	Homo sapiens	0-4	
31523846-12	2019	CONCLUSION: This preliminary study revealed that homozygote A1AT rs1303 (PiM3M3) variant is significantly higher in patients with isolated ASA and may be associated with ASA development.	Aspirin	139-142	serpin family A member 1	Homo sapiens	60-64	
31523846-12	2019	CONCLUSION: This preliminary study revealed that homozygote A1AT rs1303 (PiM3M3) variant is significantly higher in patients with isolated ASA and may be associated with ASA development.	Aspirin	170-173	serpin family A member 1	Homo sapiens	60-64	
17654043-3	2007	Twenty day treatment with 3 g of l-arginine and 75 mg of acetylsalicylic acid daily resulted in a decrease of the level of lipid peroxidation products and augmentation of alpha-1-antitrypsin activity.	Aspirin	57-77	serpin family A member 1	Homo sapiens	171-190	
19446280-1	2009	Low-dose aspirin (100 mg/day) was recently found to increase serum levels of alpha-1 protease inhibitor (A1-PI).	Aspirin	9-16	serpin family A member 1	Homo sapiens	77-103	
19446280-1	2009	Low-dose aspirin (100 mg/day) was recently found to increase serum levels of alpha-1 protease inhibitor (A1-PI).	Aspirin	9-16	serpin family A member 1	Homo sapiens	105-110	
19446280-4	2009	The previously described increase of A1-PI levels by low-dose aspirin was most likely caused by multiple endoscopies within a few days, which caused a systemic stress response.	Aspirin	62-69	serpin family A member 1	Homo sapiens	37-42	
1843906-1	1991	Alpha-1 antitrypsin (A-1-AT) levels were determined in 46 patients with aspirin sensitive asthma using the Eriksson method.	Aspirin	72-79	serpin family A member 1	Homo sapiens	21-27	
7631591-0	1995	Phenotypes of alpha-1-antitrypsin in intrinsic asthma and ASA-triad patients.	Aspirin	58-61	serpin family A member 1	Homo sapiens	14-33	
7631591-1	1995	The frequency of presence of various phenotypes of alpha 1-antitrypsin was studied in 31 patients with intrinsic asthma and 11 with ASA-Triad, and compared to a group of 200 people representative of the general population.	Aspirin	132-135	serpin family A member 1	Homo sapiens	51-70	
7631591-5	1995	Alpha-1-antitrypsin deficiency could be important in the pathogenesis of inflammatory processes and in the clinical manifestations characteristic of patients with intrinsic asthma and ASA-Triad.	Aspirin	184-187	serpin family A member 1	Homo sapiens	0-19	
1843906-3	1991	In 30 patients with aspirin asthma the phenotype of A-1-AT was determined using the Laurell method.	Aspirin	20-27	serpin family A member 1	Homo sapiens	52-58	
1843906-0	1991	[Alpha 1-antitrypsin and its phenotype in patients with asthma and aspirin hypersensitivity].	Aspirin	67-74	serpin family A member 1	Homo sapiens	1-20	
1843906-1	1991	Alpha-1 antitrypsin (A-1-AT) levels were determined in 46 patients with aspirin sensitive asthma using the Eriksson method.	Aspirin	72-79	serpin family A member 1	Homo sapiens	0-19