PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
7514480-4	1993	When Ang II (1 microM) was introduced into the dialysis perfusion medium, DOPAC output increased markedly, peaking at 219%, and returned to control with vehicle perfusion during the recovery period.	3,4-Dihydroxyphenylacetic Acid	74-79	angiotensinogen	Rattus norvegicus	5-11	
7514480-5	1993	This increase in DOPAC output with Ang II was completely blocked by co-administration of the AT1 selective antagonist, Losartan (1 microM).	3,4-Dihydroxyphenylacetic Acid	17-22	angiotensinogen	Rattus norvegicus	35-41	
1314959-8	1992	administration of angiotensin II resulted in increased (88%) DOPAC levels regardless of chronic DuP 753.	3,4-Dihydroxyphenylacetic Acid	61-66	angiotensinogen	Rattus norvegicus	18-32	
2797213-5	1989	However, 10 mumol/l angiotensin II induced an immediate, significant increase in basal DOPAC outflow which reached a maximum of 89% in the 100 mmol/l K+ and 53% in the 50 mmol/l K+ experiments.	3,4-Dihydroxyphenylacetic Acid	87-92	angiotensinogen	Rattus norvegicus	20-34