PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
9181874-2	1996	CCK in a dose of 0.75 mg had no effect on the DA level, but significantly reduced the ethanol-induced increased DOPAC and HVA level.	3,4-Dihydroxyphenylacetic Acid	112-117	cholecystokinin	Rattus norvegicus	0-3	
7478205-2	1995	CCK-8 (10 microM), administrated via a microdialysis probe, produced a significant increase in the levels of aspartate, glutamate and gamma-aminobutyric acid (GABA), but not of 3,4-dihydroxyphenylacetic acid (DOPAC), homovanillic acid (HVA), lactate and pyruvate, which were simultaneously monitored.	3,4-Dihydroxyphenylacetic Acid	209-214	cholecystokinin	Rattus norvegicus	0-3	
2351370-4	1990	Administered alone, sulfated CCK octapeptide (CCK 8 S) (5 micrograms/kg ip) and diazepam (5 mg/kg ip) were found to decrease DOPAC levels in the cortex and to induce 5-hydroxy-tryptamine accumulation in the hippocampus.	3,4-Dihydroxyphenylacetic Acid	125-130	cholecystokinin	Rattus norvegicus	29-32	
2351370-4	1990	Administered alone, sulfated CCK octapeptide (CCK 8 S) (5 micrograms/kg ip) and diazepam (5 mg/kg ip) were found to decrease DOPAC levels in the cortex and to induce 5-hydroxy-tryptamine accumulation in the hippocampus.	3,4-Dihydroxyphenylacetic Acid	125-130	cholecystokinin	Rattus norvegicus	46-49