PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
19628748-1	2010	PURPOSE: To characterize the neuroprotective mechanisms of erythropoietin (EPO) against the stress of glyoxal-advanced glycation end products (AGEs) in retinal neuronal cells.	Glyoxal	102-109	erythropoietin	Rattus norvegicus	59-73	
19628748-1	2010	PURPOSE: To characterize the neuroprotective mechanisms of erythropoietin (EPO) against the stress of glyoxal-advanced glycation end products (AGEs) in retinal neuronal cells.	Glyoxal	102-109	erythropoietin	Rattus norvegicus	75-78	
19628748-9	2010	EPO protected the retinal cells from glyoxal-AGE-induced injury in a time- and dose-dependent fashion.	Glyoxal	37-44	erythropoietin	Rattus norvegicus	0-3	
19628748-14	2010	CONCLUSIONS: These data demonstrate that exogenous EPO significantly attenuates the retinal neuronal cell death induced by glyoxal-AGEs by promoting antiapoptotic and suppressing apoptotic proteins.	Glyoxal	123-130	erythropoietin	Rattus norvegicus	51-54	
32800831-12	2020	Cellular fractionation showed EPO mitigated the VE-cadherin internalization in glyoxal-treated cells.	Glyoxal	79-86	erythropoietin	Rattus norvegicus	30-33	
31483932-4	2019	The glyoxal-treated ARPE-19 cells, incubated with EPO, soluble EPO receptor (sEPOR), Go6976, or digoxin, were studied for cell viability and barrier function.	Glyoxal	4-11	erythropoietin	Rattus norvegicus	63-66