PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
15314506-6	2004	Among the traditional NSAIDs, the mode of action of aceclofenac has been recently clarified in that the compound was shown to elicit preferential inhibition of COX-2 as a result of limited but sustained biotransformation to diclofenac.	aceclofenac	52-63	prostaglandin-endoperoxide synthase 2	Homo sapiens	160-165	
16709320-6	2006	Aceclofenac is an anti-inflammatory and analgesic drug with preferential COX-2 inhibition.	aceclofenac	0-11	prostaglandin-endoperoxide synthase 2	Homo sapiens	73-78	
34616297-1	2021	Aceclofenac (ACE), a cyclooxygenase-2 inhibitor, is the derivative of the diclofenac group that has been in use for the symptomatic treatment of systemic inflammatory autoimmune disease, rheumatoid arthritis (RA).	aceclofenac	0-11	prostaglandin-endoperoxide synthase 2	Homo sapiens	21-37	
9226412-4	1997	We also observed that part of the aceclofenac was converted into diclofenac, the cyclooxygenase-1 and cyclooxygenase-2 inhibitor, when aceclofenac was incubated with human rheumatoid synovial cells.	aceclofenac	34-45	prostaglandin-endoperoxide synthase 2	Homo sapiens	102-118	
9226412-4	1997	We also observed that part of the aceclofenac was converted into diclofenac, the cyclooxygenase-1 and cyclooxygenase-2 inhibitor, when aceclofenac was incubated with human rheumatoid synovial cells.	aceclofenac	135-146	prostaglandin-endoperoxide synthase 2	Homo sapiens	102-118