PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
16385384-8	2006	Mean plasma glucose levels at 6 h decreased from 12.4+/-1.1 (SAL) mmol/l to 10.4+/-1.1 (LSC), 8.6+/-0.6 (IB), 8.8+/-0.8 (IV) and 9.1+/-0.9 (HSC) mmol/l, p<0.0001.	Glucose	12-19	fucosyltransferase 1 (H blood group)	Homo sapiens	140-143	
24953180-7	2014	Thus, the availability of glutamine and glucose to provide fuel for nucleotide biosynthesis regulates HSC lineage commitment under conditions of metabolic stress.	Glucose	40-47	fucosyltransferase 1 (H blood group)	Homo sapiens	102-105	
16306359-12	2005	We concluded that PARP activation contributes to superoxide anion radical and peroxynitrite formation in peripheral nerve, vasa nervorum, and aorta of STZ-induced diabetic rats and high- glucose-exposed HSC.	Glucose	187-194	fucosyltransferase 1 (H blood group)	Homo sapiens	203-206	
33878034-4	2021	Using hepatocellular carcinoma (HCC) as a model, we showed that restricted glucose availability enhanced the PERK-eIF2alpha-ATF4 signaling axis to drive fucosyltransferase-1 (FUT1) transcription via direct binding of ATF4 to the FUT1 promoter.	Glucose	75-82	fucosyltransferase 1 (H blood group)	Homo sapiens	153-173	
33878034-4	2021	Using hepatocellular carcinoma (HCC) as a model, we showed that restricted glucose availability enhanced the PERK-eIF2alpha-ATF4 signaling axis to drive fucosyltransferase-1 (FUT1) transcription via direct binding of ATF4 to the FUT1 promoter.	Glucose	75-82	fucosyltransferase 1 (H blood group)	Homo sapiens	175-179	
33878034-4	2021	Using hepatocellular carcinoma (HCC) as a model, we showed that restricted glucose availability enhanced the PERK-eIF2alpha-ATF4 signaling axis to drive fucosyltransferase-1 (FUT1) transcription via direct binding of ATF4 to the FUT1 promoter.	Glucose	75-82	fucosyltransferase 1 (H blood group)	Homo sapiens	229-233	
21195127-3	2011	This study was designed to evaluate impacts of high levels of glucose on HSC activation, assess roles of the phytochemical curcumin in attenuating the glucose impacts, and elucidate underlying mechanisms.	Glucose	62-69	fucosyltransferase 1 (H blood group)	Homo sapiens	73-76	
21195127-7	2011	We observed that high levels of glucose induced cell proliferation, type I collagen production and expression of genes relevant to HSC activation, and elevated intracellular glucose levels in cultured HSCs.	Glucose	32-39	fucosyltransferase 1 (H blood group)	Homo sapiens	131-134	
21195127-11	2011	In conclusion, hyperglycemia stimulated HSC activation in vitro by increasing intracellular glucose, which was eliminated by curcumin by blocking the membrane translocation of GLUT2 and suppressing glut2 expression.	Glucose	92-99	fucosyltransferase 1 (H blood group)	Homo sapiens	40-43