PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 2207116-10 1990 The action of ALP is consistent with a mechanism in which ALP interacts with a transmembrane portion of the sugar transport molecule resulting in a competitive displacement of D-glucose or cytochalasin B from the cytosolic facing side of the transport molecule. Glucose 176-185 ATHS Homo sapiens 14-17 9185163-6 1997 While the activity of ALP of cerebral endothelial cells was maintained at the control level, we found a significant decrease in the gamma-GT activity from 3.8 +/- 1.3 to 1.09 +/- 0.3 U/mg protein after 3 h of hypoxia in the presence as well as in the absence of glucose. Glucose 262-269 ATHS Homo sapiens 22-25 2207116-7 1990 ALP acts as a competitive inhibitor of exchange L-glucose transport, of CCB binding to the glucose transporter and of D-glucose inhibition of CCB binding to the transporter. Glucose 48-57 ATHS Homo sapiens 0-3 2207116-7 1990 ALP acts as a competitive inhibitor of exchange L-glucose transport, of CCB binding to the glucose transporter and of D-glucose inhibition of CCB binding to the transporter. Glucose 118-127 ATHS Homo sapiens 0-3 2207116-10 1990 The action of ALP is consistent with a mechanism in which ALP interacts with a transmembrane portion of the sugar transport molecule resulting in a competitive displacement of D-glucose or cytochalasin B from the cytosolic facing side of the transport molecule. Glucose 176-185 ATHS Homo sapiens 58-61 34486387-6 2021 Indicators of osteogenic differentiation were all down-regulated by 40mM/L high glucose, including alkaline phosphatase(ALP) activity, runt-related transcription factor 2(RUNX2), and osteopontin(OPN) gene expression and Wnt signaling pathway. Glucose 80-87 ATHS Homo sapiens 120-123 31080819-8 2019 Results: SCAPs in 25mmol/L glucose group expressed the maximum proteins of RUNX2 and ALP as compared with those in 5, 10, and 15 mmol/L groups. Glucose 27-34 ATHS Homo sapiens 85-88 30894315-11 2019 Furthermore,EPO alleviate high glucose(30 mM) induced suppression of osteogenic differentiation ability in PDLSCs, as evidenced by the up-regulated mRNA and protein expression of Runx2 and Osterix and increased ALP activity. Glucose 31-38 ATHS Homo sapiens 211-214 31080819-10 2019 ALP assay, alizarin red staining, real-time RT-PCR, and western blot showed 25 mmol/L high glucose can obviously enhance the odonto/osteogenic capacity of SCAPs. Glucose 91-98 ATHS Homo sapiens 0-3 28525946-13 2017 From in vitro investigation, we found that the high-glucose (HG), high-lipid (HL), and beta-glycerophosphate (beta-GP) considerably increased the total calcium content, ALP activity, and expression of osteogenic markers in vascular smooth muscle cells (VSMCs). Glucose 52-59 ATHS Homo sapiens 169-172