PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 8615384-8 1996 Absorption of glucose, fructose, and lactulose was significantly inhibited by 0.1 mg x mL-1 protamine, while water absorption was decreased 41 percent by 1.0 mg x mL-1 protamine. Glucose 14-21 L1 cell adhesion molecule Mus musculus 87-91 9789584-5 1998 Assuming unchanged rate constant for loss of labelled CO2 at normo- and hyperglycaemia the oxidative metabolic rate of glucose was found to be slightly larger at combined hyperglycaemia and hypersulinemia (0.30 +/- 0.01 mmol mL-1 min-1) than at normal glucose and insulin levels (0.25 +/- 0.01 mmol mL-1 min-1). Glucose 119-126 L1 cell adhesion molecule Mus musculus 225-235 9789584-5 1998 Assuming unchanged rate constant for loss of labelled CO2 at normo- and hyperglycaemia the oxidative metabolic rate of glucose was found to be slightly larger at combined hyperglycaemia and hypersulinemia (0.30 +/- 0.01 mmol mL-1 min-1) than at normal glucose and insulin levels (0.25 +/- 0.01 mmol mL-1 min-1). Glucose 119-126 L1 cell adhesion molecule Mus musculus 299-309 9042089-9 1997 RESULTS: Fasting plasma glucose was significantly higher in HT than in NT (5.2 vs. 4.9 mmol L-1; P < 0.05). Glucose 24-31 L1 cell adhesion molecule Mus musculus 92-95 9042089-10 1997 In C fasting glucose was 5.1 mmol L-1. Glucose 13-20 L1 cell adhesion molecule Mus musculus 34-37 2333387-3 1990 Furthermore, addition of 0.1 microgram mL-1 tunicamycin to the incubation medium virtually eliminated incorporation of glucose into the protein bands but had no effect on the pattern or rate of incorporation of labelled amino acids in parallel experiments. Glucose 119-126 L1 cell adhesion molecule Mus musculus 39-43 7822430-2 1995 Lowering of the feed medium glucose concentration from 25.0 to 1.4 mmol/L resulted in a decrease of steady-state viable cell concentration from 1.9 x 10(9) to 1.0 x 10(9) L-1, whereas viability remained above 90%. Glucose 28-35 L1 cell adhesion molecule Mus musculus 171-174 34617628-7 2021 Maximum activity (2.97x10-3 U mL-1 ) was obtained at pH 5.5, 80 muL of phenylacetonitrile, and 15 g of glucose. Glucose 103-110 L1 cell adhesion molecule Mus musculus 30-34 34844168-11 2022 The fabricated sensor displayed a wide linear range (Glucose: 0.001-50 mmol L-1, AFP: 2.25 x 10-7-225 ng mL-1), impressive low limit of detection (Glucose: 1.27 x 10-7 mol L-1, AFP: 1.92 x 10-8 ng mL-1, S/N = 3) and acceptable recovery (Glucose: 94% ~ 104%, AFP: 96.04% ~ 102.29%) in practical sample. Glucose 53-60 L1 cell adhesion molecule Mus musculus 76-79 32104793-6 2020 Quantitatively, the best linear response (R2 > 0.99) was attained in the concentration range from 0.005 to 1 mg mL-1 for glucose. Glucose 121-128 L1 cell adhesion molecule Mus musculus 112-116 33376877-5 2020 With the proposed method (Benedictq), the reducing sugar glucose can be determined in a range of 0.167-10 mg mL-1, with an R 2 of 0.997 and accuracy (expressed as % of recovery) greater than 97%. Glucose 57-64 L1 cell adhesion molecule Mus musculus 109-113 34495195-5 2021 After exposure to the aqueous extract, the highest (P<0.05) glucose concentration and the lowest (P<0.05) plasma sodium level were when the fish were exposed to the S. erecta concentration of 50 microg mL-1. Glucose 60-67 L1 cell adhesion molecule Mus musculus 202-206 4084861-5 1985 The minimum inhibitory concentration for Bacteroides ruminicola 23 grown with glucose was 1 micrograms X mL-1; for B. Glucose 78-85 L1 cell adhesion molecule Mus musculus 105-109 33401694-7 2021 There was a significant interaction between time and intervention for glucose (p = 0.02), which was, on average (mean +- SD), 0.7 +- 0.7 mmol L-1 higher following high-intensity interval exercise, as compared to resistance exercise. Glucose 70-77 L1 cell adhesion molecule Mus musculus 142-145 32729490-2 2020 Under visible light irradiation, glucose was photocatalytically oxidized by A-g-C3N4 and generated H2O2 in situ, which was used as the electron acceptor for ITO/CBFeO photocathode, thus producing a high cell output response with a maximum output power of 133.5 muW cm-2 and an open circuit potential of 0.98 V. Due to the specific recognition of PSA by the aptamer and the output power decrease of the PEFC caused by the steric hindrance of the captured PSA on the TNA/A-g-C3N4, the PEFC could be used as a self-powered aptasensor for PSA with a quantitative range of 0.005-50 ng mL-1, a low detection limit of 1.3 pg mL-1 and good selectivity, and has been successfully applied for the analysis of real human serum samples with good precision of the relative standard deviation (RSD) less than 5.6% and good accuracy of the recoveries ranged from 91% to 108%. Glucose 33-40 L1 cell adhesion molecule Mus musculus 580-584 32729490-2 2020 Under visible light irradiation, glucose was photocatalytically oxidized by A-g-C3N4 and generated H2O2 in situ, which was used as the electron acceptor for ITO/CBFeO photocathode, thus producing a high cell output response with a maximum output power of 133.5 muW cm-2 and an open circuit potential of 0.98 V. Due to the specific recognition of PSA by the aptamer and the output power decrease of the PEFC caused by the steric hindrance of the captured PSA on the TNA/A-g-C3N4, the PEFC could be used as a self-powered aptasensor for PSA with a quantitative range of 0.005-50 ng mL-1, a low detection limit of 1.3 pg mL-1 and good selectivity, and has been successfully applied for the analysis of real human serum samples with good precision of the relative standard deviation (RSD) less than 5.6% and good accuracy of the recoveries ranged from 91% to 108%. Glucose 33-40 L1 cell adhesion molecule Mus musculus 618-622 32064488-6 2020 We found that within the concentration range of 0.5-2 mg mL-1, MLPs significantly inhibited glucose transport, and the inhibition rate increased with time and dose. Glucose 92-99 L1 cell adhesion molecule Mus musculus 57-61 31361066-4 2019 In vitro protein release profiles reveal that the release of protein is highly dependent on the pH or glucose concentrations, that is, less amount of protein is released at pH 7.4 or healthy blood glucose level (1 mg mL-1 glucose), while quicker release of protein occurs at pH 5.5 or diabetic blood glucose level (above 3 mg mL-1 glucose). Glucose 102-109 L1 cell adhesion molecule Mus musculus 217-221 31514873-8 2020 The high accuracy of this method was demonstrated in urine glucose measurements with a linear response over the 0.039 mg mL-1 to 10.000 mg mL-1 glucose concentration range and a 0.009 mg mL-1 detection limit. Glucose 59-66 L1 cell adhesion molecule Mus musculus 121-125 31514873-8 2020 The high accuracy of this method was demonstrated in urine glucose measurements with a linear response over the 0.039 mg mL-1 to 10.000 mg mL-1 glucose concentration range and a 0.009 mg mL-1 detection limit. Glucose 59-66 L1 cell adhesion molecule Mus musculus 139-143 31514873-8 2020 The high accuracy of this method was demonstrated in urine glucose measurements with a linear response over the 0.039 mg mL-1 to 10.000 mg mL-1 glucose concentration range and a 0.009 mg mL-1 detection limit. Glucose 59-66 L1 cell adhesion molecule Mus musculus 139-143 31514873-8 2020 The high accuracy of this method was demonstrated in urine glucose measurements with a linear response over the 0.039 mg mL-1 to 10.000 mg mL-1 glucose concentration range and a 0.009 mg mL-1 detection limit. Glucose 144-151 L1 cell adhesion molecule Mus musculus 139-143 31514873-8 2020 The high accuracy of this method was demonstrated in urine glucose measurements with a linear response over the 0.039 mg mL-1 to 10.000 mg mL-1 glucose concentration range and a 0.009 mg mL-1 detection limit. Glucose 144-151 L1 cell adhesion molecule Mus musculus 139-143 31361066-4 2019 In vitro protein release profiles reveal that the release of protein is highly dependent on the pH or glucose concentrations, that is, less amount of protein is released at pH 7.4 or healthy blood glucose level (1 mg mL-1 glucose), while quicker release of protein occurs at pH 5.5 or diabetic blood glucose level (above 3 mg mL-1 glucose). Glucose 102-109 L1 cell adhesion molecule Mus musculus 326-330 32261185-8 2013 Intriguingly, addition of 25 pg mL-1 protamine decreased the emission intensity of FA-AuNPs at 780 nm even in the presence of 1000-fold higher concentrations of Na+, K+, Ca2+, Mg2+, Fe2+, SO4 2-, Cl-, PO4 3- NO3 -, ascorbic acid, glucose interferences and bovine serum albumin interferences. Glucose 230-237 L1 cell adhesion molecule Mus musculus 32-36 29498440-5 2018 The proposed methods proved to have high sensitivities and were able to be used for determination of concentrations as low as 2.5 pg mL-1 for d-glucose and as low as 2.5 fg mL-1 for l-glucose. Glucose 142-151 L1 cell adhesion molecule Mus musculus 133-137 29498440-5 2018 The proposed methods proved to have high sensitivities and were able to be used for determination of concentrations as low as 2.5 pg mL-1 for d-glucose and as low as 2.5 fg mL-1 for l-glucose. Glucose 182-191 L1 cell adhesion molecule Mus musculus 173-177 28351626-4 2017 The amount of glucose is proportional to the phospho-p5315 concentration from 0.1 to 50 ng mL-1, the limit of detection is 50 pg mL-1 (3S/N). Glucose 14-21 L1 cell adhesion molecule Mus musculus 91-95 28351626-4 2017 The amount of glucose is proportional to the phospho-p5315 concentration from 0.1 to 50 ng mL-1, the limit of detection is 50 pg mL-1 (3S/N). Glucose 14-21 L1 cell adhesion molecule Mus musculus 129-133 27640395-7 2016 Insulin-mimetic tests on C2C12 muscle cells indicate that the complex significantly stimulated cell glucose utilization with cytotoxicity at 0.11 g L-1. Glucose 100-107 L1 cell adhesion molecule Mus musculus 148-151 32450039-7 2019 AAPs-F at 0.5 mg mL-1 significantly enhanced the glucose absorption of HepG2 cells by 24.4% (P < .05) in a dose-dependent manner at 24 h, and markedly extended the lifespan of C. elegans by 32.9% (P < 0.05) under high sugar stress conditions. Glucose 49-56 L1 cell adhesion molecule Mus musculus 17-21