PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 28132451-3 2016 Reactions of polypeptides with glucose resulting in advanced glycation end products (AGE) generation and reaction with lipid peroxidation products giving advanced lipoxidation end products (ALE) and mechanisms of the above products generation are described. Glucose 31-38 renin binding protein Homo sapiens 52-83 33204871-1 2020 Glycation of amino or guanidino groups of proteins with glucose and glucose-derived reactive aldehydes, such as alpha-hydroxyaldehydes, leads to accumulation of advanced glycation end-products (AGEs) in the body, resulting in diabetic complications and age-related pathology. Glucose 56-63 renin binding protein Homo sapiens 253-256 33204871-1 2020 Glycation of amino or guanidino groups of proteins with glucose and glucose-derived reactive aldehydes, such as alpha-hydroxyaldehydes, leads to accumulation of advanced glycation end-products (AGEs) in the body, resulting in diabetic complications and age-related pathology. Glucose 68-75 renin binding protein Homo sapiens 253-256 31981634-4 2020 The objective of the present review is to provide an update on the AGE-induced mechanisms involved in the pathophysiology of glucose and lipid metabolism, even though the unique mechanisms involved in these diseases are not well understood. Glucose 125-132 renin binding protein Homo sapiens 67-70 30179123-7 2018 AGE cannot replace HbA1c for the diagnosis and management of diabetes because there is no correlation of AGE with serum glucose, and because the half-life of protein with which glucose combines is only 14-20 days as compared to erythrocytes which have a half-life of 90-120 days. Glucose 177-184 renin binding protein Homo sapiens 0-3 28132451-3 2016 Reactions of polypeptides with glucose resulting in advanced glycation end products (AGE) generation and reaction with lipid peroxidation products giving advanced lipoxidation end products (ALE) and mechanisms of the above products generation are described. Glucose 31-38 renin binding protein Homo sapiens 85-88 15221334-2 2004 Until now AGE derived from glucose (glucose-AGE) have been mainly investigated, so we established new AGE species derived from alpha-hydroxyaldehydes and dicarbonyl compounds. Glucose 27-34 renin binding protein Homo sapiens 10-13 15221334-8 2004 The distribution of glycer-AGE differed from that of glucose-AGE, which was detected at both intracellular and extracellular sites. Glucose 53-60 renin binding protein Homo sapiens 61-64 15221334-2 2004 Until now AGE derived from glucose (glucose-AGE) have been mainly investigated, so we established new AGE species derived from alpha-hydroxyaldehydes and dicarbonyl compounds. Glucose 27-34 renin binding protein Homo sapiens 44-47 15221334-2 2004 Until now AGE derived from glucose (glucose-AGE) have been mainly investigated, so we established new AGE species derived from alpha-hydroxyaldehydes and dicarbonyl compounds. Glucose 27-34 renin binding protein Homo sapiens 44-47 15221334-2 2004 Until now AGE derived from glucose (glucose-AGE) have been mainly investigated, so we established new AGE species derived from alpha-hydroxyaldehydes and dicarbonyl compounds. Glucose 36-43 renin binding protein Homo sapiens 10-13 15221334-2 2004 Until now AGE derived from glucose (glucose-AGE) have been mainly investigated, so we established new AGE species derived from alpha-hydroxyaldehydes and dicarbonyl compounds. Glucose 36-43 renin binding protein Homo sapiens 44-47 15221334-2 2004 Until now AGE derived from glucose (glucose-AGE) have been mainly investigated, so we established new AGE species derived from alpha-hydroxyaldehydes and dicarbonyl compounds. Glucose 36-43 renin binding protein Homo sapiens 44-47 34549249-5 2021 MEASUREMENTS: Multinomial logistic regression was used to estimate odds ratios (ORs) with 95% confidence intervals (CI) for the association between age, frailty, dementia and hypoglycemia, and being prescribed insulin-only, non-insulin glucose-lowering drugs (GLDs) or combined insulin and non-insulin GLDs compared to no GLD. Glucose 236-243 renin binding protein Homo sapiens 148-151 11828884-5 2001 Cytotoxicity of the AGE-modified BSAs increased in correlation to the incubation time with glucose. Glucose 91-98 renin binding protein Homo sapiens 20-23 34880167-1 2021 PURPOSE: The purpose of this study was to determine a reference value for masticatory performance (MP) by measuring the amount of glucose extracted(AGE) from chewing gummy jelly. Glucose 130-137 renin binding protein Homo sapiens 148-151 34785935-0 2021 Association of Admission Blood Glucose Level with All-Cause Mortality According to Age in Patients with Community Acquired Pneumonia. Glucose 31-38 renin binding protein Homo sapiens 83-86 34785935-6 2021 The strength of the association of blood glucose level with 28-day mortality decreased with age (P=0.01 for the interaction) as the adjusted HRs for death were 4.48 (95% CI: 1.40-13.65; P<0.01) for middle-age patients 45-64 years and 1.52 (95% CI: 1.09-2.17; P=0.05) for elderly patients >=65 years. Glucose 41-48 renin binding protein Homo sapiens 92-95 34785935-6 2021 The strength of the association of blood glucose level with 28-day mortality decreased with age (P=0.01 for the interaction) as the adjusted HRs for death were 4.48 (95% CI: 1.40-13.65; P<0.01) for middle-age patients 45-64 years and 1.52 (95% CI: 1.09-2.17; P=0.05) for elderly patients >=65 years. Glucose 41-48 renin binding protein Homo sapiens 205-208 34060241-4 2021 Among male firefighters, there were independent and significant associations of BMI and age for the prevalence of high blood pressure, high cholesterol, high triglycerides, and high glucose. Glucose 182-189 renin binding protein Homo sapiens 88-91 33720477-0 2021 Age and aging effects on blood pressure: 15 years follow-up of Tehran lipid and glucose study. Glucose 80-87 renin binding protein Homo sapiens 0-3 35158049-7 2022 We propose that the higher AGE concentration in birds is mainly attributable to greater AGE-production due to elevated basal glucose concentrations and decreased AGE-clearance given differences in glomerular filtration rates in birds compared with mammals. Glucose 125-132 renin binding protein Homo sapiens 27-30 35158049-7 2022 We propose that the higher AGE concentration in birds is mainly attributable to greater AGE-production due to elevated basal glucose concentrations and decreased AGE-clearance given differences in glomerular filtration rates in birds compared with mammals. Glucose 125-132 renin binding protein Homo sapiens 88-91