PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 19893307-6 2010 In the tPA treatment group, admission blood glucose was higher among patients with poor functional outcome (p = 0.002). Glucose 44-51 chromosome 20 open reading frame 181 Homo sapiens 7-10 12221155-2 2002 METHOD: To analyze the relationship between admission glucose level and clinical outcomes from acute ischemic stroke, the authors performed multivariate regression analysis with the National Institute of Neurological Disorders and Stroke recombinant tissue plasminogen activator (rt-PA) Stroke Trial data. Glucose 54-61 chromosome 20 open reading frame 181 Homo sapiens 250-278 12677014-12 2003 Admission blood glucose correlated negatively with the degree of neurological improvement at 24 hours in reperfused (r=-0.43; P=0.019) but not in nonreperfused (r=-0.20; P=0.21) tPA-treated patients. Glucose 16-23 chromosome 20 open reading frame 181 Homo sapiens 178-181 12677014-14 2003 A logistic regression modeling revealed that only admission glucose value >140 mg/dL (odds ratio, 8.4; 95% CI, 1.76 to 40.02; P=0.005) emerged as an independent predictor of poor outcome despite tPA-induced recanalization. Glucose 60-67 chromosome 20 open reading frame 181 Homo sapiens 198-201 17641238-4 2007 Compared with placebo patients, patients treated with tPA who experienced SICH were older, had more severe stroke deficits, had higher serum glucose levels, and more often displayed mass effect on pretreatment imaging. Glucose 141-148 chromosome 20 open reading frame 181 Homo sapiens 54-57 1628760-0 1992 Increased expression of tissue plasminogen activator and its inhibitor and reduced fibrinolytic potential of human endothelial cells cultured in elevated glucose. Glucose 154-161 chromosome 20 open reading frame 181 Homo sapiens 24-52 18629794-9 1996 This is accompanied by changes in amino acids, glucose, lactate, and pH, which may account for the reduction of tPA productivity. Glucose 47-54 chromosome 20 open reading frame 181 Homo sapiens 112-115 8735180-7 1996 In addition, PAI-1 activity and tPA antigen were significantly correlated with blood pressure, SSPG, triglyceride, HDL-cholesterol and integrated glucose response to an oral load of 75 g glucose. Glucose 146-153 chromosome 20 open reading frame 181 Homo sapiens 32-35 8735180-7 1996 In addition, PAI-1 activity and tPA antigen were significantly correlated with blood pressure, SSPG, triglyceride, HDL-cholesterol and integrated glucose response to an oral load of 75 g glucose. Glucose 187-194 chromosome 20 open reading frame 181 Homo sapiens 32-35 33177612-7 2020 Functional activity, characterised by the measurement of nitric oxide, endothelin-1, tissue plasminogen activator and plasminogen activator inhibitor-1, was depressed by exposure to high glucose, with the reduction in nitric oxide production being the most notable. Glucose 187-194 chromosome 20 open reading frame 181 Homo sapiens 85-113