PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
12869392-13	2003	Combined with our previous studies, we conclude that trimetazidine inhibition of LC 3-KAT decreases fatty acid oxidation and stimulates glucose oxidation, resulting in an improvement in cardiac function and efficiency after ischemia.	Glucose	136-143	thiosulfate sulfurtransferase like domain containing 1	Homo sapiens	86-89	
18690865-5	2007	However, the results of current research is also supporting the concept that shifting the energy substrate preference away from fatty acid metabolism and toward glucose metabolism by 3-KAT inhibitors could be an effective adjunctive treatment in patients with heart failure, in terms of left ventricular function and glucose metabolism improvement.	Glucose	161-168	thiosulfate sulfurtransferase like domain containing 1	Homo sapiens	185-188	
18690865-5	2007	However, the results of current research is also supporting the concept that shifting the energy substrate preference away from fatty acid metabolism and toward glucose metabolism by 3-KAT inhibitors could be an effective adjunctive treatment in patients with heart failure, in terms of left ventricular function and glucose metabolism improvement.	Glucose	317-324	thiosulfate sulfurtransferase like domain containing 1	Homo sapiens	185-188	
20932648-7	2011	Trimetazidine, a 3-ketoacyl coenzyme A thiolase (3-KAT) inhibitor, shifts cardiac energy metabolism from free fatty acid oxidation to glucose oxidation by inhibiting mitochondrial long-chain 3-KAT, and is used clinically as an effective antianginal agent.	Glucose	134-141	thiosulfate sulfurtransferase like domain containing 1	Homo sapiens	193-196