PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
25749152-5	2015	reduced blood glucose levels significantly and increased the protein expressions of insulin receptor beta, insulin receptor substrate 1, protein kinase B (Akt/PKB), IKKalpha, and IkappaBalpha.	Glucose	14-21	conserved helix-loop-helix ubiquitous kinase	Mus musculus	165-173	
33860440-10	2021	While inhibiting mTOR by rapamycin or shmTOR significantly suppressed high glucose-induced activation of NF-kappaB and its regulators IKKbeta and IkappaBalpha, suggesting mTOR is the upstream regulator of NF-kappaB.	Glucose	75-82	conserved helix-loop-helix ubiquitous kinase	Mus musculus	134-141	
33864813-13	2021	The increased urine excretion of glucose and sodium was mediated by decreased renal SGLT2 expression due to inactivation of the IKKalpha/beta/NF-kappaB-induced inflammatory response.	Glucose	33-40	conserved helix-loop-helix ubiquitous kinase	Mus musculus	128-141